p57Kip2 is a repressor of Mash1 activity and neuronal differentiation in neural stem cells. (Q39826816)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19590511%20AND%20SRC:MED&resulttype=core&format=json

2 February 2020

title

p57Kip2 is a repressor of Mash1 activity and neuronal differentiation in neural stem cells (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19590511%20AND%20SRC:MED&resulttype=core&format=json

2 February 2020

1 reference

2

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2 February 2020

Erik Södersten

4

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2 February 2020

Ola Hermanson

7

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2 February 2020

author name string

B Joseph

1

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2 February 2020

P Vlachos

3

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2 February 2020

L Liu

5

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2 February 2020

A I Teixeira

6

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2 February 2020

publication date

10 July 2009

1 reference

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Cell Death & Differentiation

2 February 2020

published in

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2 February 2020

volume

16

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2 February 2020

page(s)

1256-1265

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2 February 2020

issue

9

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https://scigraph.springernature.com/pub.10.1038/cdd.2009.72

2 February 2020

exact match

0 references

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p27kip1 independently promotes neuronal differentiation and migration in the cerebral cortex

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p21 loss compromises the relative quiescence of forebrain stem cell proliferation leading to exhaustion of their proliferation capacity

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The cyclin-dependent kinase inhibitors p57 and p27 regulate neuronal migration in the developing mouse neocortex.

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Ablation of the CDK inhibitor p57Kip2 results in increased apoptosis and delayed differentiation during mouse development.

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A cell-autonomous requirement for Cip/Kip cyclin-kinase inhibitors in regulating neuronal cell cycle exit but not differentiation in the developing spinal cord

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p57(Kip2) cooperates with Nurr1 in developing dopamine cells

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Tumor-suppressor p53 is expressed in proliferating and newly formed neurons of the embryonic and postnatal rat brain: comparison with expression of the cell cycle regulators p21Waf1/Cip1, p27Kip1, p57Kip2, p16Ink4a, cyclin G1, and the proto-oncogene

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Stabilization of MyoD by direct binding to p57(Kip2)

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Suppression of cell transformation by the cyclin-dependent kinase inhibitor p57KIP2 requires binding to proliferating cell nuclear antigen

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Single factors direct the differentiation of stem cells from the fetal and adult central nervous system.

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CXXC5 is a novel BMP4-regulated modulator of Wnt signaling in neural stem cells

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The proneural determinant MASH1 regulates forebrain Dlx1/2 expression through the I12b intergenic enhancer

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p57Kip2 regulates actin dynamics by binding and translocating LIM-kinase 1 to the nucleus

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The cell cycle inhibitor p57(Kip2) promotes cell death via the mitochondrial apoptotic pathway.

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BMPs inhibit neurogenesis by a mechanism involving degradation of a transcription factor

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The Notch signaling pathway controls the size of the ocular lens by directly suppressing p57Kip2 expression

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Loss of intestinal crypt progenitor cells owing to inactivation of both Notch1 and Notch2 is accompanied by derepression of CDK inhibitors p27Kip1 and p57Kip2.

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The cyclin-dependent kinase inhibitor p57kip2 is a negative regulator of Schwann cell differentiation and in vitro myelination.

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Early cortical precursors do not undergo LIF-mediated astrocytic differentiation

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inferred from DOI database lookup

7 January 2021

based on heuristic

Pituitary adenylate cyclase activating polypeptide anti-mitogenic signaling in cerebral cortical progenitors is regulated by p57Kip2-dependent CDK2 activity.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.72

inferred from DOI database lookup

7 January 2021

based on heuristic

p27Xic1, a Cdk inhibitor, promotes the determination of glial cells in Xenopus retina

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.72