Overexpression of BMI-1 correlates with drug resistance in B-cell lymphoma cells through the stabilization of survivin expression. (Q39697369)

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14 February 2020

title

Overexpression of BMI-1 correlates with drug resistance in B-cell lymphoma cells through the stabilization of survivin expression (English)

1 reference

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14 February 2020

1 reference

1 reference

3

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14 February 2020

8

Masaharu Hoshi

1 reference

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14 February 2020

7

Akira Sakai

1 reference

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14 February 2020

Joyeeta Bhattacharyya

1

object named as

Joyeeta Bhattacharyya

1 reference

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14 February 2020

Akiro Kimura

10

object named as

Akiro Kimura

1 reference

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14 February 2020

Keichiro Mihara

2

object named as

Keichiro Mihara

1 reference

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14 February 2020

author name string

Shin'ichiro Yasunaga

4

1 reference

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14 February 2020

Yoshifumi Takei

5

1 reference

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14 February 2020

Kazuyoshi Yanagihara

6

1 reference

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14 February 2020

Yoshihiro Takihara

9

1 reference

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14 February 2020

language of work or name

1 reference

20 November 2011

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14 February 2020

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14 February 2020

volume

103

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14 February 2020

issue

1

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14 February 2020

page(s)

34-41

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Deregulated expression of Polycomb-group oncogenes in human malignant lymphomas and epithelial tumors

14 February 2020

cites work

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Novel zinc finger gene implicated as myc collaborator by retrovirally accelerated lymphomagenesis in E mu-myc transgenic mice.

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Bmi1, stem cells, and senescence regulation

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BMI-1 expression is enhanced through transcriptional and posttranscriptional regulation during the progression of chronic myeloid leukemia

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The polycomb group protein Bmi-1 represses the tumor suppressor PTEN and induces epithelial-mesenchymal transition in human nasopharyngeal epithelial cells

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The polycomb group BMI1 gene is a molecular marker for predicting prognosis of chronic myeloid leukemia.

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Cancer cells survive with survivin

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Validating survivin as a cancer therapeutic target

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Adenoviral infection of survivin antisense sensitizes prostate cancer cells to etoposide in vivo

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Survivin expression and resistance to anticancer treatments: perspectives for new therapeutic interventions.

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Alternative reading frames of the INK4a tumor suppressor gene encode two unrelated proteins capable of inducing cell cycle arrest

21 January 2018

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The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus

21 January 2018

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Bmi-1 Regulation of INK4A-ARF Is a Downstream Requirement for Transformation of Hematopoietic Progenitors by E2a-Pbx1

21 January 2018

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Combination of cyclooxygenase-2 inhibitors and oxaliplatin increases the growth inhibition and death in human colon cancer cells

21 January 2018

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Oxaliplatin, a potent inhibitor of survivin, enhances paclitaxel-induced apoptosis and mitotic catastrophe in colon cancer cells

21 January 2018

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Effective immunochemotherapy for aggressive non-Hodgkin's lymphoma

21 January 2018

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Regulation of survivin function by Hsp90

21 January 2018

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Modulation of Akt kinase activity by binding to Hsp90

21 January 2018

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Expression of Bmi-1 in epidermis enhances cell survival by altering cell cycle regulatory protein expression and inhibiting apoptosis

21 January 2018

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21 January 2018

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A novel anti-apoptosis gene, survivin, expressed in cancer and lymphoma

21 January 2018

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