Toll-like receptor 3 mediates expression of clusterin/apolipoprotein J in vascular smooth muscle cells stimulated with RNA released from necrotic cells. (Q39670059)

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scientific article published on 5 August 2010

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English	Toll-like receptor 3 mediates expression of clusterin/apolipoprotein J in vascular smooth muscle cells stimulated with RNA released from necrotic cells.	scientific article published on 5 August 2010

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9 February 2020

Toll-like receptor 3 mediates expression of clusterin/apolipoprotein J in vascular smooth muscle cells stimulated with RNA released from necrotic cells (English)

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toll-like receptor

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Christian Lehmann

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M Baiersdörfer

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M Schwarz

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K Seehafer

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A Heit

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H Wagner

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C J Kirschning

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C Koch-Brandt

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Experimental Cell Research

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3489-3500

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9 February 2020

Human clusterin gene expression is confined to surviving cells during in vitro programmed cell death

1 reference

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Clusterin: physiologic and pathophysiologic considerations.

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Apolipoprotein J/clusterin limits the severity of murine autoimmune myocarditis

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Inhibition of post-ischemic brain injury by clusterin overexpression

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Sustained astrocytic clusterin expression improves remodeling after brain ischemia.

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Clusterin is a secreted mammalian chaperone

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Clusterin/apolipoprotein J in human aging and cancer

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Clusterin, an abundant serum factor, is a possible negative regulator of MT6-MMP/MMP-25 produced by neutrophils.

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Essential requirement of apolipoprotein J (clusterin) signaling for IkappaB expression and regulation of NF-kappaB activity.

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Multiple receptors mediate apoJ-dependent clearance of cellular debris into nonprofessional phagocytes

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Apoptotic cell debris and phosphatidylserine-containing lipid vesicles induce apolipoprotein J (clusterin) gene expression in vital fibroblasts

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Apolipoprotein J/clusterin is induced in vascular smooth muscle cells after vascular injury.

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Distribution and synthesis of apolipoprotein J in the atherosclerotic aorta

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Apolipoprotein J (clusterin) induces cholesterol export from macrophage-foam cells: a potential anti-atherogenic function?

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Clusterin (Apo J) regulates vascular smooth muscle cell differentiation in vitro

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Clusterin regulates vascular smooth muscle cell nodule formation and migration

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Apolipoprotein J inhibits the migration, adhesion, and proliferation of vascular smooth muscle cells

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Innate immune recognition

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Innate immune sensing and its roots: the story of endotoxin

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Pathogen recognition with Toll-like receptors

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Regulation of phagosome maturation by signals from toll-like receptors.

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Toll-like receptors induce a phagocytic gene program through p38.

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Toll-like receptors stimulate human neutrophil function

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Toll-like receptor-4 is expressed by macrophages in murine and human lipid-rich atherosclerotic plaques and upregulated by oxidized LDL.

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Expression of toll-like receptors in human atherosclerotic lesions: a possible pathway for plaque activation

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Endogenous ligands of Toll-like receptors: implications for regulating inflammatory and immune responses.

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Endogenous TLR ligands and autoimmunity

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Multiple roles of Toll-like receptor signaling in atherosclerosis

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Proinflammatory phenotype of vascular smooth muscle cells: role of efficient Toll-like receptor 4 signaling.

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Toll-like receptor 2 mediates persistent chemokine release by Chlamydia pneumoniae-infected vascular smooth muscle cells

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Role of Toll-like receptors 2 and 4 in the induction of cyclooxygenase-2 in vascular smooth muscle

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Toll-like receptor 3 signaling evokes a proinflammatory and proliferative phenotype in human vascular smooth muscle cells.

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Toll-like receptor signalling

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An essential role of the NF-kappa B/Toll-like receptor pathway in induction of inflammatory and tissue-repair gene expression by necrotic cells

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10.1016/J.YEXCR.2010.07.022

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9 February 2020

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