High-level expression of Mastermind-like 2 contributes to aberrant activation of the NOTCH signaling pathway in human lymphomas. (Q39625552)

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17 January 2020

title

High-level expression of Mastermind-like 2 contributes to aberrant activation of the NOTCH signaling pathway in human lymphomas (English)

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17 January 2020

8

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author name string

K Köchert

1

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17 January 2020

K Ullrich

2

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17 January 2020

S Kreher

3

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17 January 2020

J C Aster

4

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17 January 2020

M Kitagawa

5

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17 January 2020

K Jöhrens

6

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17 January 2020

I Anagnostopoulos

7

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17 January 2020

B Lamprecht

9

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17 January 2020

U Zimber-Strobl

10

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17 January 2020

H Stein

11

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17 January 2020

M Janz

12

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17 January 2020

B Dörken

13

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17 January 2020

S Mathas

14

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language of work or name

English

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publication date

29 November 2010

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17 January 2020

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17 January 2020

volume

30

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17 January 2020

issue

15

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17 January 2020

page(s)

1831-1840

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https://scigraph.springernature.com/pub.10.1038/onc.2010.544

17 January 2020

exact match

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cites work

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TAN-1, the human homolog of the Drosophila Notch gene, is broken by chromosomal translocations in T lymphoblastic neoplasms

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Mastermind mediates chromatin-specific transcription and turnover of the Notch enhancer complex

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Notch 1 activation in the molecular pathogenesis of T-cell acute lymphoblastic leukaemia

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HES and HERP families: multiple effectors of the Notch signaling pathway

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Classical Hodgkin lymphoma is characterized by high constitutive expression of activating transcription factor 3 (ATF3), which promotes viability of Hodgkin/Reed-Sternberg cells

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Characterization of a high-molecular-weight Notch complex in the nucleus of Notch(ic)-transformed RKE cells and in a human T-cell leukemia cell line

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The mastermind-like 1 (MAML1) co-activator regulates constitutive NF-kappaB signaling and cell survival

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Aberrant expression of Notch1 interferes with the B-lymphoid phenotype of neoplastic B cells in classical Hodgkin lymphoma.

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Activated Notch1 signaling promotes tumor cell proliferation and survival in Hodgkin and anaplastic large cell lymphoma.

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Interleukin 13 is secreted by and stimulates the growth of Hodgkin and Reed-Sternberg cells

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Oncogenesis of T-ALL and nonmalignant consequences of overexpressing intracellular NOTCH1

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Loss of bHLH transcription factor E2A activity in primary effusion lymphoma confers resistance to apoptosis.

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Identification of new human mastermind proteins defines a family that consists of positive regulators for notch signaling

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Intrinsic inhibition of transcription factor E2A by HLH proteins ABF-1 and Id2 mediates reprogramming of neoplastic B cells in Hodgkin lymphoma

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Direct inhibition of the NOTCH transcription factor complex

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Structural basis for cooperativity in recruitment of MAML coactivators to Notch transcription complexes

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