Influenza A induces the major secreted airway mucin MUC5AC in a protease-EGFR-extracellular regulated kinase-Sp1-dependent pathway. (Q39387104)

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24 January 2020

title

Influenza A induces the major secreted airway mucin MUC5AC in a protease-EGFR-extracellular regulated kinase-Sp1-dependent pathway (English)

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24 January 2020

4

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David J. Thornton

7

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24 January 2020

Mustapha Si-Tahar

8

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Jean-Michel Sallenave

11

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9

Lhousseine Touqui

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24 January 2020

author name string

Diane Barbier

1

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24 January 2020

Ignacio Garcia-Verdugo

2

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24 January 2020

Julien Pothlichet

3

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Delphyne Descamps

5

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Karine Rousseau

6

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24 January 2020

Michel Chignard

10

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publication date

1 March 2012

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American Journal of Respiratory Cell and Molecular Biology

24 January 2020

published in

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volume

47

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issue

2

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page(s)

149-157

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Confronting the avian influenza threat: vaccine development for a potential pandemic.

24 January 2020

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Induction of MUC2 and MUC5AC mucins by factors of the epidermal growth factor (EGF) family is mediated by EGF receptor/Ras/Raf/extracellular signal-regulated kinase cascade and Sp1.

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PMA induces the MUC5AC respiratory mucin in human bronchial epithelial cells, via PKC, EGF/TGF-alpha, Ras/Raf, MEK, ERK and Sp1-dependent mechanisms.

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Protein kinase C delta regulates airway mucin secretion via phosphorylation of MARCKS protein

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Rhinovirus-induced major airway mucin production involves a novel TLR3-EGFR-dependent pathway

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Rhinovirus induces MUC5AC in a human infection model and in vitro via NF-κB and EGFR pathways.

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Identifiers

DOI

10.1165/RCMB.2011-0405OC

1 reference

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24 January 2020

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