Dacarbazine-mediated upregulation of NKG2D ligands on tumor cells activates NK and CD8 T cells and restrains melanoma growth (Q39284974)

10 September 2017

Dacarbazine-mediated upregulation of NKG2D ligands on tumor cells activates NK and CD8 T cells and restrains melanoma growth (English)

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10 September 2017

2

Cédric Rébé

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François Ghiringhelli

8

François Ghiringhelli

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Lionel Apetoh

7

Lionel Apetoh

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Mélanie Bruchard

5

Mélanie Bruchard

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Grégoire Mignot

9

Grégoire Mignot

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author name string

Alice Hervieu

1

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10 September 2017

Frédérique Végran

3

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10 September 2017

Fanny Chalmin

4

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10 September 2017

Pierre Vabres

6

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10 September 2017

language of work or name

English

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publication date

6 September 2012

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Journal of Investigative Dermatology

10 September 2017

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10 September 2017

133

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10 September 2017

2

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10 September 2017

499-508

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https://scigraph.springernature.com/pub.10.1038/jid.2012.273

10 September 2017

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cites work

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Immunogenicity of anthracyclines: moving towards more personalized medicine

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Restoration of antitumor immunity through selective inhibition of myeloid derived suppressor cells by anticancer therapies

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Expression of adhesion molecules and ligands for activating and costimulatory receptors involved in cell-mediated cytotoxicity in a large panel of human melanoma cell lines

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Retinoic acid early inducible genes define a ligand family for the activating NKG2D receptor in mice

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Ectopic expression of retinoic acid early inducible-1 gene (RAE-1) permits natural killer cell-mediated rejection of a MHC class I-bearing tumor in vivo

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Effect of NKG2D ligand expression on host immune responses

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Ligands for the murine NKG2D receptor: expression by tumor cells and activation of NK cells and macrophages.

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Rae1 and H60 ligands of the NKG2D receptor stimulate tumour immunity

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Experimental mouse tumour models: what can be learnt about human cancer immunology?

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Chemotherapy-induced genotoxic stress promotes sensitivity to natural killer cell cytotoxicity by enabling missing-self recognition

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Unique Functional Status of Natural Killer Cells in Metastatic Stage IV Melanoma Patients and Its Modulation by Chemotherapy

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Induction of NKG2D ligands by gamma radiation and tumor necrosis factor-alpha may participate in the tissue damage during acute graft-versus-host disease

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The DNA damage pathway regulates innate immune system ligands of the NKG2D receptor

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Combination Chemotherapy in Disseminated Melanoma and Other Solid Tumors in Adults

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Activation of the NLRP3 inflammasome in dendritic cells induces IL-1beta-dependent adaptive immunity against tumors

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CD4+CD25+ regulatory T cells suppress tumor immunity but are sensitive to cyclophosphamide which allows immunotherapy of established tumors to be curative

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Improved Survival with Ipilimumab in Patients with Metastatic Melanoma

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Immunotherapy of malignant melanoma

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Ipilimumab plus dacarbazine in melanoma

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Increase of NKG2D ligands and sensitivity to NK cell-mediated cytotoxicity of tumor cells by heat shock and ionizing radiation.

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Radiation-induced IFN-gamma production within the tumor microenvironment influences antitumor immunity

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Treatments for metastatic melanoma: synthesis of evidence from randomized trials

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Regulation of ligands for the activating receptor NKG2D.

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Stress-regulated targeting of the NKG2D ligand Mult1 by a membrane-associated RING-CH family E3 ligase

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Calreticulin exposure dictates the immunogenicity of cancer cell death

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Dacarbazine treatment before peptide vaccination enlarges T-cell repertoire diversity of melan-a-specific, tumor-reactive CTL in melanoma patients.

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7 January 2021

Differential clinical significance of individual NKG2D ligands in melanoma: soluble ULBP2 as an indicator of poor prognosis superior to S100B.

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Role of NKG2D in tumor cell lysis mediated by human NK cells: cooperation with natural cytotoxicity receptors and capability of recognizing tumors of nonepithelial origin

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7 January 2021

Melanoma cells inhibit natural killer cell function by modulating the expression of activating receptors and cytolytic activity

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What is the role of cytotoxic T lymphocyte-associated antigen 4 blockade in patients with metastatic melanoma?

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Ipilimumab plus dacarbazine for previously untreated metastatic melanoma

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Soluble NKG2D ligands: prevalence, release, and functional impact.

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ATM-ATR-dependent up-regulation of DNAM-1 and NKG2D ligands on multiple myeloma cells by therapeutic agents results in enhanced NK-cell susceptibility and is associated with a senescent phenotype

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ULBPs, human ligands of the NKG2D receptor, stimulate tumor immunity with enhancement by IL-15.

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Gemcitabine selectively eliminates splenic Gr-1+/CD11b+ myeloid suppressor cells in tumor-bearing animals and enhances antitumor immune activity

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Dual therapeutic efficacy of vinblastine as a unique chemotherapeutic agent capable of inducing dendritic cell maturation

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CTLA-4 blockade: therapeutic potential in cancer treatments

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Combination immunotherapy of B16 melanoma using anti-cytotoxic T lymphocyte-associated antigen 4 (CTLA-4) and granulocyte/macrophage colony-stimulating factor (GM-CSF)-producing vaccines induces rejection of subcutaneous and metastatic tumors accomp

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5-Fluorouracil selectively kills tumor-associated myeloid-derived suppressor cells resulting in enhanced T cell-dependent antitumor immunity

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7 January 2021

The emerging role of CTLA4 as a cell-extrinsic regulator of T cell responses

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Cancer immunotherapy using a bispecific NK receptor fusion protein that engages both T cells and tumor cells

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Immunological aspects of cancer chemotherapy

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Interferon-gamma treatment of B16 melanoma cells: opposing effects for non-adaptive and adaptive immune defense and its reflection by metastatic spread

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7 January 2021

Identifiers

DOI

10.1038/JID.2012.273

1 reference

Dimensions Publication ID

10 September 2017

1005047084

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Fatcat ID

release_y44hgl5gtrbutayndcqltxmrwi

0 references

1 reference