Tumor-derived CXCL8 signaling augments stroma-derived CCL2-promoted proliferation and CXCL12-mediated invasion of PTEN-deficient prostate cancer cells. (Q38980564)

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English	Tumor-derived CXCL8 signaling augments stroma-derived CCL2-promoted proliferation and CXCL12-mediated invasion of PTEN-deficient prostate cancer cells.	scientific article

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21 March 2020

Tumor-derived CXCL8 signaling augments stroma-derived CCL2-promoted proliferation and CXCL12-mediated invasion of PTEN-deficient prostate cancer cells (English)

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21 March 2020

prostate cancer

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4

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21 March 2020

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Pamela J Maxwell

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21 March 2020

Jessica Neisen

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21 March 2020

Johanna Messenger

3

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21 March 2020

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1 July 2014

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21 March 2020

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21 March 2020

5

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21 March 2020

13

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21 March 2020

4895-4908

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21 March 2020

describes a project that uses

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11 June 2022

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Targeting CXCL12 from FAP-expressing carcinoma-associated fibroblasts synergizes with anti-PD-L1 immunotherapy in pancreatic cancer

1 reference

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Loss of PTEN is associated with aggressive behavior in ERG-positive prostate cancer

1 reference

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Advancing precision medicine for prostate cancer through genomics

1 reference

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Potentiation of inflammatory CXCL8 signalling sustains cell survival in PTEN-deficient prostate carcinoma.

1 reference

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9 September 2017

Loss of PTEN expression is associated with increased risk of recurrence after prostatectomy for clinically localized prostate cancer

1 reference

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Constitutive and treatment-induced CXCL8-signalling selectively modulates the efficacy of anti-metabolite therapeutics in metastatic prostate cancer

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CXCR1 and CXCR2 silencing modulates CXCL8-dependent endothelial cell proliferation, migration and capillary-like structure formation

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CCL2 as an important mediator of prostate cancer growth in vivo through the regulation of macrophage infiltration

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CCL2 is a potent regulator of prostate cancer cell migration and proliferation

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Pten dose dictates cancer progression in the prostate

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4148108

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ETS factors reprogram the androgen receptor cistrome and prime prostate tumorigenesis in response to PTEN loss

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Aberrant ERG expression cooperates with loss of PTEN to promote cancer progression in the prostate

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4148108

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Chemotherapy-induced CXC-chemokine/CXC-chemokine receptor signaling in metastatic prostate cancer cells confers resistance to oxaliplatin through potentiation of nuclear factor-kappaB transcription and evasion of apoptosis.

1 reference

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Nonapical and cytoplasmic expression of interleukin-8, CXCR1, and CXCR2 correlates with cell proliferation and microvessel density in prostate cancer

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Is PTEN loss associated with clinical outcome measures in human prostate cancer?

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10.18632/ONCOTARGET.2052

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21 March 2020

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21 March 2020

1 reference

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21 March 2020

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