Involvement of NLRP10 in IL-1α induction of oral epithelial cells by periodontal pathogens (Q38647155)

5 September 2017

title

Involvement of NLRP10 in IL-1α induction of oral epithelial cells by periodontal pathogens (English)

1 reference

NLR family pyrin domain containing 10

5 September 2017

main subject

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positive regulation of defense response to bacterium

GOA release 2020-03-11

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GOA release 2020-03-11

author name string

Seok-Joo Lee

series ordinal

1

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5 September 2017

Bong-Kyu Choi

series ordinal

2

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5 September 2017

publication date

1 January 2017

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5 September 2017

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5 September 2017

page(s)

1753425917722610

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Functions of NOD-Like Receptors in Human Diseases

5 September 2017

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Activation of NLRP3 and AIM2 inflammasomes by Porphyromonas gingivalis infection

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Contradictory roles of Porphyromonas gingivalis gingipains in caspase-1 activation

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Aggregatibacter actinomycetemcomitans cytolethal distending toxin activates the NLRP3 inflammasome in human macrophages, leading to the release of proinflammatory cytokines

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Expression and regulation of the NALP3 inflammasome complex in periodontal diseases.

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Fusobacterium nucleatum infection of gingival epithelial cells leads to NLRP3 inflammasome-dependent secretion of IL-1β and the danger signals ASC and HMGB1.

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Aggregatibacter actinomycetemcomitans targets NLRP3 and NLRP6 inflammasome expression in human mononuclear leukocytes.

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Aggregatibacter actinomycetemcomitans outer membrane vesicles are internalized in human host cells and trigger NOD1- and NOD2-dependent NF-κB activation

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Inflammasome-activated gasdermin D causes pyroptosis by forming membrane pores

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Interleukin-1 signal transduction

21 January 2018

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IL-1 plays a critical role in oral, but not dermal, wound healing

21 January 2018

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The interleukin-1 genotype as a severity factor in adult periodontal disease

21 January 2018

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Relationship between interleukin 1α levels in the gingival crevicular fluid in health and in inflammatory periodontal disease and periodontal inflamed surface area: A correlative study

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HIV-1 reactivation induced by the periodontal pathogens Fusobacterium nucleatum and Porphyromonas gingivalis involves Toll-like receptor 2 [corrected] and 9 activation in monocytes/macrophages

21 January 2018

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Tea polyphenols inhibit the activation of NF-κB and the secretion of cytokines and matrix metalloproteinases by macrophages stimulated with Fusobacterium nucleatum

21 January 2018

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NOD1 and NOD2 mediate sensing of periodontal pathogens

21 January 2018

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Regulation of NLRP3 and AIM2 inflammasome gene expression levels in gingival fibroblasts by oral biofilms

21 January 2018

1 reference

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Toll-like receptor 2 and NALP2 mediate induction of human beta-defensins by fusobacterium nucleatum in gingival epithelial cells

21 January 2018

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Regulation of NOD-like receptors and inflammasome activation in cerebral endothelial cells

21 January 2018

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The NLRP3 inflammasome is released as a particulate danger signal that amplifies the inflammatory response

21 January 2018

1 reference

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Quantitative Determination of IL-1α-Induced IL-8 mRNA Levels in Cultured Human Keratinocytes, Dermal Fibroblasts, Endothelial Cells, and Monocytes

21 January 2018

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21 January 2018

Identifiers

DOI

10.1177/1753425917722610

1 reference

5 September 2017

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