Hepatitis C virus NS5A and subgenomic replicon activate NF-kappaB via tyrosine phosphorylation of IkappaBalpha and its degradation by calpain protease (Q38351589)

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English	Hepatitis C virus NS5A and subgenomic replicon activate NF-kappaB via tyrosine phosphorylation of IkappaBalpha and its degradation by calpain protease	scientific article

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scholarly article

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Europe PubMed Central

2 September 2017

retracted paper

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Dataset for "Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine"

Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine

Hepatitis C virus NS5A and subgenomic replicon activate NF-kappaB via tyrosine phosphorylation of IkappaBalpha and its degradation by calpain protease (English)

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Europe PubMed Central

2 September 2017

phosphorylation

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Hepatitis C virus

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based on heuristic

inferred from title

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based on heuristic

inferred from title

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based on heuristic

inferred from title

5

object named as

Aleem Siddiqui

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Europe PubMed Central

2 September 2017

author name string

Gulam Waris

1

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Europe PubMed Central

2 September 2017

Antonia Livolsi

2

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Europe PubMed Central

2 September 2017

Veronique Imbert

3

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Europe PubMed Central

2 September 2017

Jean-Francois Peyron

4

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Europe PubMed Central

2 September 2017

language of work or name

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publication date

7 August 2003

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Europe PubMed Central

2 September 2017

number of pages

10

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based on heuristic

inferred from page(s)

Journal of Biological Chemistry

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Europe PubMed Central

2 September 2017

278

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Europe PubMed Central

2 September 2017

40778-40787

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Europe PubMed Central

2 September 2017

42

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Europe PubMed Central

2 September 2017

Molecular biology of the hepatitis C viruses: implications for diagnosis, development and control of viral disease

1 reference

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Hepatitis B and C viral infections in patients with hepatocellular carcinoma

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Hepatitis C virus infection is associated with the development of hepatocellular carcinoma

1 reference

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Replication of subgenomic hepatitis C virus RNAs in a hepatoma cell line

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Selectable subgenomic and genome-length dicistronic RNAs derived from an infectious molecular clone of the HCV-N strain of hepatitis C virus replicate efficiently in cultured Huh7 cells

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Persistent and transient replication of full-length hepatitis C virus genomes in cell culture

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Replication of hepatitis C virus

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The nonstructural NS5A protein of hepatitis C virus: an expanding, multifunctional role in enhancing hepatitis C virus pathogenesis

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Hepatitis C virus RNA polymerase and NS5A complex with a SNARE-like protein

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Hepatitis C virus (HCV) NS5A binds RNA-dependent RNA polymerase (RdRP) NS5B and modulates RNA-dependent RNA polymerase activity

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Hepatitis C virus NS5A physically associates with p53 and regulates p21/waf1 gene expression in a p53-dependent manner

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Human hepatitis C virus NS5A protein alters intracellular calcium levels, induces oxidative stress, and activates STAT-3 and NF-kappa B.

1 reference

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I kappa B: a specific inhibitor of the NF-kappa B transcription factor

1 reference

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NF-kappaB at the crossroads of life and death

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Methyl-CpG-binding proteins. Targeting specific gene repression.

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Crucial role of the amino-terminal tyrosine residue 42 and the carboxyl-terminal PEST domain of I kappa B alpha in NF-kappa B activation by an oxidative stress

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Genetic evidence for differential coupling of Syk family kinases to the T-cell receptor: reconstitution studies in a ZAP-70-deficient Jurkat T-cell line

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Cell biology. Apoptosis--the calcium connection.

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The inhibition of mitochondrial calcium transport by lanthanides and ruthenium red.

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v-Src-induced modulation of the calpain-calpastatin proteolytic system regulates transformation.

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Regulation of calpain and calpastatin in differentiating myoblasts: mRNA levels, protein synthesis and stability

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Cutting to the chase: calpain proteases in cell motility

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Ionizing radiation and short wavelength UV activate NF-kappaB through two distinct mechanisms

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Tumor necrosis factor-alpha-inducible IkappaBalpha proteolysis mediated by cytosolic m-calpain. A mechanism parallel to the ubiquitin-proteasome pathway for nuclear factor-kappab activation

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Novel IkappaB alpha proteolytic pathway in WEHI231 immature B cells

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Calpain: a cytosolic proteinase active at the membranes

1 reference

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Calpastatin is up-regulated in response to hypoxia and is a suicide substrate to calpain after neonatal cerebral hypoxia-ischemia.

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Oxygen-bridged dinuclear ruthenium amine complex specifically inhibits Ca2+ uptake into mitochondria in vitro and in situ in single cardiac myocytes

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Calpain activation in plasma membrane bleb formation during tert-butyl hydroperoxide-induced rat hepatocyte injury.

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The role of the C-terminal domain of I kappa B alpha in protein degradation and stabilization

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Casein kinase II phosphorylates I kappa B alpha at S-283, S-289, S-293, and T-291 and is required for its degradation

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Constitutive phosphorylation of IkappaBalpha by casein kinase II occurs preferentially at serine 293: requirement for degradation of free IkappaBalpha

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Barriers come down

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Identifiers

10.1074/JBC.M303248200

1 reference

Europe PubMed Central

2 September 2017

1 reference

Europe PubMed Central

2 September 2017

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