Oxidative stress activates the plasminogen activator inhibitor type 1 (PAI-1) promoter through an AP-1 response element and cooperates with insulin for additive effects on PAI-1 transcription (Q38342702)

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English	Oxidative stress activates the plasminogen activator inhibitor type 1 (PAI-1) promoter through an AP-1 response element and cooperates with insulin for additive effects on PAI-1 transcription	scientific article

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scholarly article

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Europe PubMed Central

2 September 2017

Oxidative stress activates the plasminogen activator inhibitor type 1 (PAI-1) promoter through an AP-1 response element and cooperates with insulin for additive effects on PAI-1 transcription (English)

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Europe PubMed Central

2 September 2017

1 reference

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author name string

Anthony I Vulin

1

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Europe PubMed Central

2 September 2017

Frederick M Stanley

2

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Europe PubMed Central

2 September 2017

language of work or name

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publication date

5 April 2004

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Europe PubMed Central

2 September 2017

number of pages

7

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Journal of Biological Chemistry

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Europe PubMed Central

2 September 2017

279

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Europe PubMed Central

2 September 2017

24

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Europe PubMed Central

2 September 2017

25172-25178

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Europe PubMed Central

2 September 2017

Induction of plasminogen activator inhibitor type-1 (PAI-1) by proinsulin and insulin in vivo.

1 reference

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Augmentation of the synthesis of plasminogen activator inhibitor type-1 by precursors of insulin. A potential risk factor for vascular disease.

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Regulation of proteolytic enzymes and inhibitors in two smooth muscle cell phenotypes

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Characterization of the activation of latent TGF-beta by co-cultures of endothelial cells and pericytes or smooth muscle cells: a self-regulating system

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Distribution and regulation of plasminogen activator inhibitor-1 in murine adipose tissue in vivo. Induction by tumor necrosis factor-alpha and lipopolysaccharide

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Effect of thrombin on release of plasminogen activator inhibitor-1 from cultured primate arterial smooth muscle cells.

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A Forkhead/winged helix-related transcription factor mediates insulin-increased plasminogen activator inhibitor-1 gene transcription.

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Diabetes mellitus and free radicals. Free radicals, transition metals and oxidative stress in the aetiology of diabetes mellitus and complications

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Diabetic endothelial dysfunction: role of reactive oxygen and nitrogen species production and poly(ADP-ribose) polymerase activation.

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Insulin-like growth factor-1-mediated neuroprotection against oxidative stress is associated with activation of nuclear factor kappaB.

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c-Jun/AP-1, but not NF-kappa B, is a mediator for oxidant-initiated apoptosis in glomerular mesangial cells

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H2O2 and tumor necrosis factor-alpha induce differential binding of the redox-responsive transcription factors AP-1 and NF-kappaB to the interleukin-8 promoter in endothelial and epithelial cells

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Role of oxidants and antioxidants in the induction of AP-1, NF-kappaB, and glutathione S-transferase gene expression

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Improved primer design for PCR-based, site-directed mutagenesis

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Transcriptional regulation of prolactin gene expression by thyroid hormone--alternate suppression and stimulation in different GH cell lines

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Depletion of L-3,5,3'-Triiodothyronine and L-Thyroxine in Euthyroid Calf Serum for Use in Cell Culture Studies of the Action of Thyroid Hormone*

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NF kappa B and AP-1 mediate transcriptional responses to oxidative stress in skeletal muscle cells

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JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

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JNK-independent activation of c-Jun during neuronal apoptosis induced by multiple DNA-damaging agents

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Activator protein 1 (AP-1)- and nuclear factor kappaB (NF-kappaB)-dependent transcriptional events in carcinogenesis

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Glucose upregulates plasminogen activator inhibitor-1 gene expression in vascular smooth muscle cells

1 reference

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21 January 2018

Transcription factor decoy for activator protein-1 (AP-1) inhibits high glucose- and angiotensin II-induced type 1 plasminogen activator inhibitor (PAI-1) gene expression in cultured human vascular smooth muscle cells

1 reference

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21 January 2018

Sp1 sites mediate activation of the plasminogen activator inhibitor-1 promoter by glucose in vascular smooth muscle cells

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Requirement for p38 and p44/p42 mitogen-activated protein kinases in RAGE-mediated nuclear factor-kappaB transcriptional activation and cytokine secretion

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Activation of transcription factor NF-kappaB and p38 mitogen-activated protein kinase is mediated by distinct and separate stress effector pathways

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Selective activation of p38 MAPK cascade and mitotic arrest caused by low level oxidative stress

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UV induces phosphorylation of protein kinase B (Akt) at Ser-473 and Thr-308 in mouse epidermal C1 41 cells through hydrogen peroxide

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Transcriptional regulation of heme oxygenase-1 gene expression by MAP kinases of the JNK and p38 pathways in primary cultures of rat hepatocytes

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Involvement of c-Jun N-terminal kinase in oxidative stress-mediated suppression of insulin gene expression

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On the role of c-Jun in the induction of PAI-1 gene expression by phorbol ester, serum, and IL-1alpha in HepG2 cells

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PKC delta mediates ionizing radiation-induced activation of c-Jun NH(2)-terminal kinase through MKK7 in human thyroid cells

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Oxidant-induced inhibition of myocardial calcium-independent phospholipase A2.

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Delayed oxidant-induced cell death involves activation of phospholipase A2.

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Cytokine expression in hepatocytes: role of oxidant stress

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Plasminogen-activator inhibitor type 1 and coronary artery disease

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Accelerated skin wound healing in plasminogen activator inhibitor-1-deficient mice

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Activation of AP-1 and Nuclear Factor-κB Transcription Factors Is Involved in Hydrogen Peroxide-Induced Apoptotic Cell Death of Olligodendrocytes

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21 January 2018

Identifiers

10.1074/JBC.M403184200

1 reference

Europe PubMed Central

2 September 2017

1 reference

Europe PubMed Central

2 September 2017

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