Loss of one HuD allele on chromosome #1p selects for amplification of the N-myc proto-oncogene in human neuroblastoma cells (Q38318980)

2 September 2017

Loss of one HuD allele on chromosome #1p selects for amplification of the N-myc proto-oncogene in human neuroblastoma cells (English)

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2 September 2017

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Grandinetti KB

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2 September 2017

Spengler BA

series ordinal

2

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2 September 2017

Biedler JL

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3

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2 September 2017

Ross RA

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4

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2 September 2017

language of work or name

English

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publication date

1 February 2006

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2 September 2017

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2 September 2017

25

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5

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2 September 2017

706-712

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1209095

2 September 2017

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Amplification of N-myc in untreated human neuroblastomas correlates with advanced disease stage

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Evidence for two tumour suppressor loci on chromosomal bands 1p35-36 involved in neuroblastoma: one probably imprinted, another associated with N-myc amplification

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Allelic loss of chromosome 1p as a predictor of unfavorable outcome in patients with neuroblastoma

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Binding of a 40-kDa protein to the N-myc 3'-untranslated region correlates with enhanced N-myc expression in human neuroblastoma

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Deletion screening of the Duchenne muscular dystrophy locus via multiplex DNA amplification

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Embryonic lethality in mice homozygous for a targeted disruption of the N-myc gene

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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The Myc oncoprotein: a critical evaluation of transactivation and target gene regulation

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Loss of heterozygosity for the short arm of chromosome 1 in human neuroblastomas: correlation with N-myc amplification

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Localization of human elav-like neuronal protein 1 (Hel-N1) on chromosome 9p21 by chromosome microdissection polymerase chain reaction and fluorescence in situ hybridization

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Multiplex PCR: critical parameters and step-by-step protocol

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Identification and characterization of the NMYC gene product in human neuroblastoma cells by monoclonal antibodies with defined specificities

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Mammalian homologs of Drosophila ELAV localized to a neuronal subset can bind in vitro to the 3' UTR of mRNA encoding the Id transcriptional repressor

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ELAV, a Drosophila neuron-specific protein, mediates the generation of an alternatively spliced neural protein isoform.

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HuD, a neuronal-specific RNA-binding protein, is a putative regulator of N-myc pre-mRNA processing/stability in malignant human neuroblasts

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Clinical relevance of tumor cell ploidy and N-myc gene amplification in childhood neuroblastoma: a Pediatric Oncology Group study

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HuD, a neuronal-specific RNA-binding protein, increases the in vivo stability of MYCN RNA.

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Molecular biology of neuroblastoma

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Gene amplification-associated cytogenetic aberrations and protein changes in vincristine-resistant Chinese hamster, mouse, and human cells

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Molecular genetics of neuroblastoma and the implications for clinical management: a review of the MSKCC experience

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N-myc proto-oncogene expression during organogenesis in the developing mouse as revealed by in situ hybridization

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Mapping of the gene coding for a paraneoplastic encephalomyelitis antigen (HuD) to human chromosome site 1p34

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Two distinct deleted regions on the short arm of chromosome I in neuroblastoma

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A novel intron element operates posttranscriptionally To regulate human N-myc expression

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HuD, a paraneoplastic encephalomyelitis antigen, contains RNA-binding domains and is homologous to Elav and Sex-lethal

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7 January 2021

There may be two tumor suppressor genes on chromosome arm 1p closely associated with biologically distinct subtypes of neuroblastoma

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In vivo elimination of acentric double minutes containing amplified MYCN from neuroblastoma tumor cells through the formation of micronuclei

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Human neuroblastoma cell lines that express N-myc without gene amplification.

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Targeted expression of MYCN causes neuroblastoma in transgenic mice

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Identifiers

DOI

10.1038/SJ.ONC.1209095

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Dimensions Publication ID

2 September 2017

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1 reference