The physiopathological role of IL-33: new highlights in bone biology and a proposed role in periodontal disease. (Q38201456)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24692848%20AND%20SRC:MED&resulttype=core&format=json

12 March 2020

review article

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title

The physiopathological role of IL-33: new highlights in bone biology and a proposed role in periodontal disease (English)

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12 March 2020

5 references

21 March 2019

3 October 2019

6 October 2019

11 October 2019

20 November 2019

human musculoskeletal system

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6 October 2019

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11 October 2019

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20 November 2019

Ana Paula Lima Oliveira

2

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12 March 2020

Marcelo Silva

5

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12 March 2020

author name string

Felipe Andrés Cordero da Luz

1

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12 March 2020

Daniella Borges

3

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12 March 2020

Paula Cristina Brígido

4

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language of work or name

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18 February 2014

6 references

21 March 2019

3 October 2019

6 October 2019

11 October 2019

20 November 2019

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24692848%20AND%20SRC:MED&resulttype=core&format=json

Mediators of Inflammation

12 March 2020

published in

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3 October 2019

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11 October 2019

20 November 2019

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volume

2014

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12 March 2020

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342410

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Creative Commons Attribution 3.0 Unported

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cites work

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IL-33 is processed into mature bioactive forms by neutrophil elastase and cathepsin G

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Human basophils and eosinophils are the direct target leukocytes of the novel IL-1 family member IL-33.

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Molecular mimicry between IL-33 and KSHV for attachment to chromatin through the H2A-H2B acidic pocket

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IL-33 exacerbates antigen-induced arthritis by activating mast cells

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A novel IL-1 family cytokine, IL-33, potently activates human eosinophils

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Regulation of bone remodeling by the central and peripheral nervous system

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IL-33, a potent inducer of adaptive immunity to intestinal nematodes

6 September 2017

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B lymphocytes and the skeleton

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IL-1 receptor accessory protein and ST2 comprise the IL-33 receptor complex

6 September 2017

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Soluble ST2 blocks interleukin-33 signaling in allergic airway inflammation

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IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines

6 September 2017

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14 September 2017

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31 August 2018

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IL-33 shifts the balance from osteoclast to alternatively activated macrophage differentiation and protects from TNF-alpha-mediated bone loss.

31 August 2018

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31 August 2018

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IL-33 receptor (T1/ST2) signalling is necessary to prevent the development of encephalitis in mice infected with Toxoplasma gondii.

31 August 2018

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IL-33 amplifies both Th1- and Th2-type responses through its activity on human basophils, allergen-reactive Th2 cells, iNKT and NK cells.

31 August 2018

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31 August 2018

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12 December 2020

An IL-1 family member requires caspase-1 processing and signals through the ST2 receptor

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12 December 2020

Increased levels of interleukin-33 associated with bone erosion and interstitial lung diseases in patients with rheumatoid arthritis

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12 December 2020

Transgenic over-expression of interleukin-33 in osteoblasts results in decreased osteoclastogenesis

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12 December 2020

Cutting edge: The ST2 ligand IL-33 potently activates and drives maturation of human mast cells

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12 December 2020

Identifiers

DOI

10.1155/2014/342410

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24692848%20AND%20SRC:MED&resulttype=core&format=json

12 March 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24692848%20AND%20SRC:MED&resulttype=core&format=json

12 March 2020

PubMed publication ID

24692848

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:24692848%20AND%20SRC:MED&resulttype=core&format=json