Redox regulation of cardiac hypertrophy (Q38188338)

30 August 2017

Redox regulation of cardiac hypertrophy (English)

1 reference

30 August 2017

3

1 reference

30 August 2017

Can M Sag

1

1 reference

PubMed ID

24530760

30 August 2017

Celio X C Santos

series ordinal

2

1 reference

30 August 2017

11 February 2014

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Journal of Molecular and Cellular Cardiology

30 August 2017

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30 August 2017

73

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30 August 2017

103-111

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Cardiac remodeling--concepts and clinical implications: a consensus paper from an international forum on cardiac remodeling. Behalf of an International Forum on Cardiac Remodeling

30 August 2017

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Physiological myocardial hypertrophy: how and why?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Controversies in ventricular remodelling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Redox signaling in cardiac physiology and pathology.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nitric oxide and peroxynitrite in health and disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Inactivation of peroxiredoxin I by phosphorylation allows localized H(2)O(2) accumulation for cell signaling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Reductive stress linked to small HSPs, G6PD, and Nrf2 pathways in heart disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Activation of NADPH oxidase during progression of cardiac hypertrophy to failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Oxidant stress from nitric oxide synthase-3 uncoupling stimulates cardiac pathologic remodeling from chronic pressure load

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Mitochondrial oxidative stress mediates angiotensin II-induced cardiac hypertrophy and Galphaq overexpression-induced heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Xanthine oxidase inhibition with febuxostat attenuates systolic overload-induced left ventricular hypertrophy and dysfunction in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Mitochondria and reactive oxygen species

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Mitochondria as a therapeutic target in heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Mitochondrial electron transport complex I is a potential source of oxygen free radicals in the failing myocardium

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Essential role for mitochondrial thioredoxin reductase in hematopoiesis, heart development, and heart function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Thioredoxin reductase-2 is essential for keeping low levels of H(2)O(2) emission from isolated heart mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Overexpression of catalase targeted to mitochondria attenuates murine cardiac aging.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

The sarcoplasmic reticulum luminal thiol oxidase ERO1 regulates cardiomyocyte excitation-coupled calcium release and response to hemodynamic load

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

NADPH oxidases in cardiovascular disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nox proteins in signal transduction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Endoplasmic reticulum stress and Nox-mediated reactive oxygen species signaling in the peripheral vasculature: potential role in hypertension

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Pathological cardiac hypertrophy alters intracellular targeting of phosphodiesterase type 5 from nitric oxide synthase-3 to natriuretic peptide signaling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

S-glutathionylation uncouples eNOS and regulates its cellular and vascular function.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Monoamine oxidase B prompts mitochondrial and cardiac dysfunction in pressure overloaded hearts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Knockdown of cytochrome P450 2E1 inhibits oxidative stress and apoptosis in the cTnT(R141W) dilated cardiomyopathy transgenic mice.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Mammalian xanthine oxidoreductase - mechanism of transition from xanthine dehydrogenase to xanthine oxidase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Xanthine oxidoreductase inhibition causes reverse remodeling in rats with dilated cardiomyopathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Regulation of cardiac hypertrophy by intracellular signalling pathways

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

In search of new therapeutic targets and strategies for heart failure: recent advances in basic science

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Redox signaling in cardiac myocytes.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

A redox-dependent pathway for regulating class II HDACs and cardiac hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Neurohormonal regulation of cardiac histone deacetylase 5 nuclear localization by phosphorylation-dependent and phosphorylation-independent mechanisms.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Pivotal role of a gp91(phox)-containing NADPH oxidase in angiotensin II-induced cardiac hypertrophy in mice.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Requirement of Rac1 in the development of cardiac hypertrophy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

NADPH oxidase-derived superoxide anion mediates angiotensin II-induced cardiac hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nox2-containing NADPH oxidase and Akt activation play a key role in angiotensin II-induced cardiomyocyte hypertrophy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Apoptosis signal-regulating kinase 1 plays a pivotal role in angiotensin II-induced cardiac hypertrophy and remodeling.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Involvement of nuclear factor-kappaB and apoptosis signal-regulating kinase 1 in G-protein-coupled receptor agonist-induced cardiomyocyte hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Reactive oxygen species in mechanical stress-induced cardiac hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Alpha-adrenergic receptor-stimulated hypertrophy in adult rat ventricular myocytes is mediated via thioredoxin-1-sensitive oxidative modification of thiols on Ras.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Strain-stimulated hypertrophy in cardiac myocytes is mediated by reactive oxygen species-dependent Ras S-glutathiolation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Contrasting roles of NADPH oxidase isoforms in pressure-overload versus angiotensin II-induced cardiac hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Activation of NADPH oxidase 4 in the endoplasmic reticulum promotes cardiomyocyte autophagy and survival during energy stress through the protein kinase RNA-activated-like endoplasmic reticulum kinase/eukaryotic initiation factor 2α/activating trans

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

NADPH oxidase-4 mediates protection against chronic load-induced stress in mouse hearts by enhancing angiogenesis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

NADPH-oxidase 4 protects against kidney fibrosis during chronic renal injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nox4 is a protective reactive oxygen species generating vascular NADPH oxidase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nox4 regulates Nrf2 and glutathione redox in cardiomyocytes in vivo.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

NADPH oxidases in heart failure: poachers or gamekeepers?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Redox regulation of the hypoxia-inducible factor

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Cysteine redox sensor in PKGIa enables oxidant-induced activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Redox regulation of sodium and calcium handling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Oxidant-induced activation of type I protein kinase A is mediated by RI subunit interprotein disulfide bond formation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Oxidation of CaMKII determines the cardiotoxic effects of aldosterone.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Oxidized Ca(2+)/calmodulin-dependent protein kinase II triggers atrial fibrillation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Diabetes increases mortality after myocardial infarction by oxidizing CaMKII

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Differential cardiac remodeling in preload versus afterload

7 January 2021

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https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Redox regulation of cardiac calcium channels and transporters

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Redox modification of ryanodine receptors contributes to sarcoplasmic reticulum Ca2+ leak in chronic heart failure.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nitroxyl triggers Ca2+ release from skeletal and cardiac sarcoplasmic reticulum by oxidizing ryanodine receptors

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nitroxyl improves cellular heart function by directly enhancing cardiac sarcoplasmic reticulum Ca2+ cycling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Ionizing radiation regulates cardiac Ca handling via increased ROS and activated CaMKII.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

CaMKII-dependent SR Ca leak contributes to doxorubicin-induced impaired Ca handling in isolated cardiac myocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

X-ROS signaling: rapid mechano-chemo transduction in heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Tachycardia increases NADPH oxidase activity and RyR2 S-glutathionylation in ventricular muscle

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

HNO enhances SERCA2a activity and cardiomyocyte function by promoting redox-dependent phospholamban oligomerization

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Hydroxyl radical inhibits sarcoplasmic reticulum Ca(2+)-ATPase function by direct attack on the ATP binding site

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Oxidation of multiple methionine residues impairs rapid sodium channel inactivation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Redox signaling and cardiac sarcomeres

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Modulation of titin-based stiffness by disulfide bonding in the cardiac titin N2-B unique sequence

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nitroxyl-mediated disulfide bond formation between cardiac myofilament cysteines enhances contractile function.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

p53-induced inhibition of Hif-1 causes cardiac dysfunction during pressure overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Activation of a HIF1alpha-PPARgamma axis underlies the integration of glycolytic and lipid anabolic pathways in pathologic cardiac hypertrophy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

NADPH oxidase 4 promotes endothelial angiogenesis through endothelial nitric oxide synthase activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

NO triggers RGS4 degradation to coordinate angiogenesis and cardiomyocyte growth

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Molecular mechanisms that control interstitial fibrosis in the pressure-overloaded heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Myofibroblast-mediated mechanisms of pathological remodelling of the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Pivotal role of cardiomyocyte TGF-β signaling in the murine pathological response to sustained pressure overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Endothelial-to-mesenchymal transition contributes to cardiac fibrosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Aldosterone mediates angiotensin II-induced interstitial cardiac fibrosis via a Nox2-containing NADPH oxidase.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Involvement of the nicotinamide adenosine dinucleotide phosphate oxidase isoform Nox2 in cardiac contractile dysfunction occurring in response to pressure overload.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Involvement of Nox2 NADPH oxidase in adverse cardiac remodeling after myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Critical role of the NAD(P)H oxidase subunit p47phox for left ventricular remodeling/dysfunction and survival after myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Critical role of apoptosis signal-regulating kinase 1 in aldosterone/salt-induced cardiac inflammation and fibrosis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Endothelial NADPH oxidase-2 promotes interstitial cardiac fibrosis and diastolic dysfunction through proinflammatory effects and endothelial-mesenchymal transition.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Allopurinol attenuates left ventricular remodeling and dysfunction after experimental myocardial infarction: a new action for an old drug?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Oxidative stress and cardiac repair/remodeling following infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Increased Nox2 expression in human cardiomyocytes after acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Beneficial effects of long-term use of the antioxidant probucol in heart failure in the rat.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Beneficial effects of exogenous tetrahydrobiopterin on left ventricular remodeling after myocardial infarction in rats: the possible role of oxidative stress caused by uncoupled endothelial nitric oxide synthase.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nitric oxide and cardiac function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Ryanodine receptor leak mediated by caspase-8 activation leads to left ventricular injury after myocardial ischemia-reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Effects of long-term vitamin E supplementation on cardiovascular events and cancer: a randomized controlled trial

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Residual NADPH oxidase and survival in chronic granulomatous disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Reversal of cardiac hypertrophy and fibrosis from pressure overload by tetrahydrobiopterin: efficacy of recoupling nitric oxide synthase as a therapeutic strategy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Systemic and vascular oxidation limits the efficacy of oral tetrahydrobiopterin treatment in patients with coronary artery disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Mitochondrial targeted antioxidant Peptide ameliorates hypertensive cardiomyopathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Mitochondria-Targeted Antioxidant MitoQ 10 Improves Endothelial Function and Attenuates Cardiac Hypertrophy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Thioredoxin-1 gene therapy enhances angiogenic signaling and reduces ventricular remodeling in infarcted myocardium of diabetic rats

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

Nrf2 deficiency prevents reductive stress-induced hypertrophic cardiomyopathy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

CaMKII in the cardiovascular system: sensing redox states.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.02.002

10.1016/J.YJMCC.2014.02.002

7 January 2021

Identifiers

DOI

1 reference

30 August 2017

1 reference

PubMed ID

24530760