Checkpoints in productive and latent infections with herpes simplex virus 1: conceptualization of the issues (Q37952586)

25 August 2017

Checkpoints in productive and latent infections with herpes simplex virus 1: conceptualization of the issues (English)

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25 August 2017

1

Bernard Roizman

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25 August 2017

2

Guoying Zhou

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25 August 2017

Te Du

3

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25 August 2017

4 November 2011

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25 August 2017

Journal of NeuroVirology

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25 August 2017

17

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25 August 2017

512-517

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25 August 2017

6

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https://scigraph.springernature.com/pub.10.1007/s13365-011-0058-x

25 August 2017

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Neither LAT nor open reading frame P mutations increase expression of spliced or intron-containing ICP0 transcripts in mouse ganglia latently infected with herpes simplex virus

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Herpes simplex virus-infected cell protein 0 blocks the silencing of viral DNA by dissociating histone deacetylases from the CoREST-REST complex

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The two functions of herpes simplex virus 1 ICP0, inhibition of silencing by the CoREST/REST/HDAC complex and degradation of PML, are executed in tandem

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A herpes simplex virus transcript abundant in latently infected neurons is dispensable for establishment of the latent state

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Circadian CLOCK histone acetyl transferase localizes at ND10 nuclear bodies and enables herpes simplex virus gene expression

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The histone acetyltransferase CLOCK is an essential component of the herpes simplex virus 1 transcriptome that includes TFIID, ICP4, ICP27, and ICP22.

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Herpes simplex virus 1 alpha regulatory protein ICP0 functionally interacts with cellular transcription factor BMAL1.

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In vivo changes in the patterns of chromatin structure associated with the latent herpes simplex virus type 1 genome in mouse trigeminal ganglia can be detected at early times after butyrate treatment

21 January 2018

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The checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complex

21 January 2018

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An inquiry into the mechanisms of herpes simplex virus latency

21 January 2018

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An acutely and latently expressed herpes simplex virus 2 viral microRNA inhibits expression of ICP34.5, a viral neurovirulence factor

21 January 2018

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Herpesviral latency-associated transcript gene promotes assembly of heterochromatin on viral lytic-gene promoters in latent infection

21 January 2018

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Structural basis for CoREST-dependent demethylation of nucleosomes by the human LSD1 histone demethylase.

21 January 2018

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The CoREST/REST repressor is both necessary and inimical for expression of herpes simplex virus genes

21 January 2018

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LSD1 demethylates repressive histone marks to promote androgen-receptor-dependent transcription

21 January 2018

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12 December 2020

An essential role for CoREST in nucleosomal histone 3 lysine 4 demethylation

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12 December 2020

MicroRNAs expressed by herpes simplex virus 1 during latent infection regulate viral mRNAs

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12 December 2020

Evidence that two latency-associated transcripts of herpes simplex virus type 1 are nonlinear

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12 December 2020

10.1007/S13365-011-0058-X

Identifiers

DOI

1 reference

25 August 2017

1 reference

PubMed ID

22052379