Systems bioenergetics of creatine kinase networks: physiological roles of creatine and phosphocreatine in regulation of cardiac cell function. (Q37851388)

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scientific article published on 10 March 2011

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English	Systems bioenergetics of creatine kinase networks: physiological roles of creatine and phosphocreatine in regulation of cardiac cell function.	scientific article published on 10 March 2011

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Systems bioenergetics of creatine kinase networks: physiological roles of creatine and phosphocreatine in regulation of cardiac cell function (English)

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Marcela Gonzalez-Granillo

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Vladimir Chekulayev

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R Guzun

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N Timohhina

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A V Kuznetsov

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T Kaambre

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V A Saks

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10 March 2011

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1333-1348

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https://scigraph.springernature.com/pub.10.1007/s00726-011-0854-x

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Creatine and its potential therapeutic value for targeting cellular energy impairment in neurodegenerative diseases

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Compartmentalized energy transfer in cardiomyocytes: use of mathematical modeling for analysis of in vivo regulation of respiration

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Functions and effects of creatine in the central nervous system.

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Different kinetics of the regulation of respiration in permeabilized cardiomyocytes and in HL-1 cardiac cells. Importance of cell structure/organization for respiration regulation

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Mitochondrial dynamics in heart cells: very low amplitude high frequency fluctuations in adult cardiomyocytes and flow motion in non beating Hl-1 cells.

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The creatine-creatine phosphate energy shuttle

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Transport of energy in muscle: the phosphorylcreatine shuttle

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Inhibition of the mitochondrial permeability transition by creatine kinase substrates. Requirement for microcompartmentation.

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Regulation of respiration controlled by mitochondrial creatine kinase in permeabilized cardiac cells in situ. Importance of system level properties

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On the hypothesis that the failing heart is energy starved: lessons learned from the metabolism of ATP and creatine

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Energy metabolism in heart failure and remodelling

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Creatine kinase of heart mitochondria: changes in its kinetic properties induced by coupling to oxidative phosphorylation.

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Multimodal functional cardiac MRI in creatine kinase-deficient mice reveals subtle abnormalities in myocardial perfusion and mechanics

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