Mitochondrial biogenesis and fragmentation as regulators of muscle protein degradation (Q37802264)

23 August 2017

Mitochondrial biogenesis and fragmentation as regulators of muscle protein degradation (English)

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23 August 2017

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Vanina Romanello

1

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PubMed ID

20967516

23 August 2017

Marco Sandri

series ordinal

2

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23 August 2017

1 December 2010

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Current Hypertension Reports

23 August 2017

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23 August 2017

12

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23 August 2017

6

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23 August 2017

433-439

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https://scigraph.springernature.com/pub.10.1007/s11906-010-0157-8

23 August 2017

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Autophagy is required to maintain muscle mass.

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Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression

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Therapeutic approaches for muscle wasting disorders

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The changing shape of mitochondrial apoptosis

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Mitochondrial fission and remodelling contributes to muscle atrophy.

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Peroxisome proliferator-activated receptor gamma coactivator 1alpha or 1beta overexpression inhibits muscle protein degradation, induction of ubiquitin ligases, and disuse atrophy

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Mitochondrial function and apoptotic susceptibility in aging skeletal muscle

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Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

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12 December 2020

Properties of skeletal muscle mitochondria isolated from subsarcolemmal and intermyofibrillar regions.

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12 December 2020

10.1007/S11906-010-0157-8

Identifiers

DOI

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23 August 2017

1 reference

PubMed ID

20967516