Identification of a novel BET bromodomain inhibitor-sensitive, gene regulatory circuit that controls Rituximab response and tumour growth in aggressive lymphoid cancers. (Q37621025)

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scientific article published on 04 July 2013

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English	Identification of a novel BET bromodomain inhibitor-sensitive, gene regulatory circuit that controls Rituximab response and tumour growth in aggressive lymphoid cancers.	scientific article published on 04 July 2013

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scholarly article

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4 March 2020

Identification of a novel BET bromodomain inhibitor-sensitive, gene regulatory circuit that controls Rituximab response and tumour growth in aggressive lymphoid cancers (English)

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4 March 2020

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Sophie Rousseaux

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Patricia Betton

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Florence Combes

12

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author name string

Anouk Emadali

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Juliana Bruder-Costa

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Samuel Duley

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Sieme Hamaidia

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Alexandra Debernardi

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Dominique Leroux

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Benoit Bernay

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Sylvie Kieffer-Jaquinod

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Elena Ferri

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Charles E McKenna

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Carlo Petosa

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Christophe Bruley

16

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Mary B Callanan

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Jérôme Garin

17

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4 March 2020

publication date

4 July 2013

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EMBO Molecular Medicine

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5

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1180-1195

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4 March 2020

8

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4 March 2020

Ectopic activation of germline and placental genes identifies aggressive metastasis-prone lung cancers

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Genetic heterogeneity of diffuse large B-cell lymphoma

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The cell-cycle regulator c-Myc is essential for the formation and maintenance of germinal centers

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Patients with diffuse large B-cell lymphoma of germinal center origin with BCL2 translocations have poor outcome, irrespective of MYC status: a report from an International DLBCL rituximab-CHOP Consortium Program Study

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BET bromodomain inhibition targets both c-Myc and IL7R in high-risk acute lymphoblastic leukemia

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MYC+ diffuse large B-cell lymphoma is not salvaged by classical R-ICE or R-DHAP followed by BEAM plus autologous stem cell transplantation

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Targeting MYC dependence in cancer by inhibiting BET bromodomains

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BET bromodomain inhibition as a therapeutic strategy to target c-Myc

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Anti-CD47 antibody synergizes with rituximab to promote phagocytosis and eradicate non-Hodgkin lymphoma

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Quantitative proteomics analysis of the secretory pathway

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12 December 2020

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Reprogramming of HeLa cells upon DNMT3L overexpression mimics carcinogenesis

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10.1002/EMMM.201202034

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4 March 2020

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4 March 2020

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4 March 2020

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