Role of reactive oxygen species-elicited apoptosis in the pathophysiology of mitochondrial and neurodegenerative diseases associated with mitochondrial DNA mutations. (Q37576767)

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scientific article published on August 2009

Language	Label	Description	Also known as
English	Role of reactive oxygen species-elicited apoptosis in the pathophysiology of mitochondrial and neurodegenerative diseases associated with mitochondrial DNA mutations.	scientific article published on August 2009

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scholarly article

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Role of reactive oxygen species-elicited apoptosis in the pathophysiology of mitochondrial and neurodegenerative diseases associated with mitochondrial DNA mutations (English)

1 reference

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5 February 2020

apoptotic process

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mitochondrial DNA

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1 reference

based on heuristic

inferred from title

pathophysiology

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based on heuristic

inferred from title

neurodegeneration

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based on heuristic

inferred from title

reactive oxygen species

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based on heuristic

inferred from title

3

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author name string

Chun-Yi Liu

1

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Cheng-Feng Lee

2

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publication date

1 August 2009

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Journal of the Formosan Medical Association

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108

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8

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599-611

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5 February 2020

describes a project that uses

mitochondrial DNA

1 reference

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A mitochondrial paradigm of metabolic and degenerative diseases, aging, and cancer: a dawn for evolutionary medicine

1 reference

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Somatic mtDNA mutations and aging--facts and fancies

1 reference

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7 January 2021

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The mitochondrial electron transport and oxidative phosphorylation system

1 reference

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7 January 2021

based on heuristic

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Mitochondria in homeostasis of reactive oxygen species in cell, tissues, and organism

1 reference

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7 January 2021

based on heuristic

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Role of mitochondria in oxidative stress and ageing

1 reference

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7 January 2021

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Generation of superoxide anion by the NADH dehydrogenase of bovine heart mitochondria

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Titrating the effects of mitochondrial complex I impairment in the cell physiology

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based on heuristic

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Mitochondria as the central control point of apoptosis

1 reference

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7 January 2021

based on heuristic

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

1 reference

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7 January 2021

based on heuristic

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

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7 January 2021

based on heuristic

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Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

1 reference

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7 January 2021

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Apoptosis-inducing factor (AIF): a novel caspase-independent death effector released from mitochondria

1 reference

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7 January 2021

based on heuristic

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Role of the mitochondrial membrane permeability transition in cell death

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based on heuristic

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

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7 January 2021

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Reactive oxygen species generated by the mitochondrial respiratory chain affect the complex III activity via cardiolipin peroxidation in beef-heart submitochondrial particles

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The effect of reactive oxygen species generated from the mitochondrial electron transport chain on the cytochrome c oxidase activity and on the cardiolipin content in bovine heart submitochondrial particles

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Reactive oxygen species generated from the mitochondrial electron transport chain induce cytochrome c dissociation from beef-heart submitochondrial particles via cardiolipin peroxidation. Possible role in the apoptosis

1 reference

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Self-regulatory role of 4-hydroxynonenal in signaling for stress-induced programmed cell death.

1 reference

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Normal oxidative damage to mitochondrial and nuclear DNA is extensive

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Mitochondrial DNA mutations in human disease

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What causes mitochondrial DNA deletions in human cells?

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The 4977 bp deletion of mitochondrial DNA in human skeletal muscle, heart and different areas of the brain: a useful biomarker or more?

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7 January 2021

based on heuristic

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Spontaneous Kearns-Sayre/chronic external ophthalmoplegia plus syndrome associated with a mitochondrial DNA deletion: a slip-replication model and metabolic therapy

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A direct repeat is a hotspot for large-scale deletion of human mitochondrial DNA.

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7 January 2021

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Singlet oxygen mediates the UVA-induced generation of the photoaging-associated mitochondrial common deletion.

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Concurrent increase of oxidative DNA damage and lipid peroxidation together with mitochondrial DNA mutation in human lung tissues during aging--smoking enhances oxidative stress on the aged tissues

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Analysis of Common Deletion (CD) and a novel deletion of mitochondrial DNA induced by ionizing radiation

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based on heuristic

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Age-associated oxygen damage and mutations in mitochondrial DNA in human hearts

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Deleted 4977-bp mitochondrial DNA mutation is associated with sporadic amyotrophic lateral sclerosis: a hospital-based case-control study.

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Brain Cytochrome Oxidase in Alzheimer's Disease

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7 January 2021

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Elevated levels of the Kearns-Sayre syndrome mitochondrial DNA deletion in temporal cortex of Alzheimer's patients

1 reference

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7 January 2021

based on heuristic

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Marked changes in mitochondrial DNA deletion levels in Alzheimer brains

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7 January 2021

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High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease

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7 January 2021

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Mitochondrial DNA mutations and aging.

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7 January 2021

based on heuristic

inferred from DOI database lookup

Biological markers of oxidative stress in mitochondrial myopathies with progressive external ophthalmoplegia

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Apoptosis-related changes in skeletal muscles of patients with mitochondrial diseases

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Apoptosis in mitochondrial encephalomyopathies with mitochondrial DNA mutations: a potential pathogenic mechanism

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Apoptosis is suspended in muscle of mitochondrial encephalomyopathies

1 reference

https://api.crossref.org/works/10.1016%2FS0929-6646%2809%2960380-6

7 January 2021

based on heuristic

inferred from DOI database lookup

Apoptosis in mitochondrial myopathies is linked to mitochondrial proliferation

1 reference

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7 January 2021

based on heuristic

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Oxidative stress and neurotoxicity

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7 January 2021

based on heuristic

inferred from DOI database lookup

A cell biological perspective on mitochondrial dysfunction in Parkinson disease and other neurodegenerative diseases

1 reference

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7 January 2021

based on heuristic

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Mitochondrial biology and oxidative stress in Parkinson disease pathogenesis

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Histochemical detection of apoptosis in Parkinson's disease.

1 reference

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7 January 2021

based on heuristic

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Is Bax a mitochondrial mediator in apoptotic death of dopaminergic neurons in Parkinson's disease?

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Increased caspase 3 and Bax immunoreactivity accompany nuclear GAPDH translocation and neuronal apoptosis in Parkinson's disease

1 reference

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7 January 2021

based on heuristic

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Caspase-8 is an effector in apoptotic death of dopaminergic neurons in Parkinson's disease, but pathway inhibition results in neuronal necrosis.

1 reference

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Bax ablation prevents dopaminergic neurodegeneration in the 1-methyl- 4-phenyl-1,2,3,6-tetrahydropyridine mouse model of Parkinson's disease

1 reference

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7 January 2021

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inferred from DOI database lookup

Injection of mitochondria into human cells leads to a rapid replacement of the endogenous mitochondrial DNA.

1 reference

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7 January 2021

based on heuristic

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Human cells lacking mtDNA: repopulation with exogenous mitochondria by complementation

1 reference

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7 January 2021

based on heuristic

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Mitochondrial DNA mutation and depletion increase the susceptibility of human cells to apoptosis

1 reference

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based on heuristic

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1 reference

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inferred from DOI database lookup

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1 reference

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based on heuristic

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1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Mitochondrial DNA deletions sensitize cells to apoptosis at low heteroplasmy levels

1 reference

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7 January 2021

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1 reference

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1 reference

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1 reference

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1 reference

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Maternally-inherited Leigh syndrome-related mutations bolster mitochondrial-mediated apoptosis

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1 reference

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Mitochondria dysfunction of Alzheimer's disease cybrids enhances Abeta toxicity

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7 January 2021

based on heuristic

inferred from DOI database lookup

Premature ageing in mice expressing defective mitochondrial DNA polymerase

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1 reference

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1 reference

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Effects of coenzyme Q10 in early Parkinson disease: evidence of slowing of the functional decline

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