Multi-drug strategies are necessary to inhibit the synergistic mechanism causing tissue damage and organ failure in post infectious sequelae (Q36885542)

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scientific article published on January 1999

Language	Label	Description	Also known as
English	Multi-drug strategies are necessary to inhibit the synergistic mechanism causing tissue damage and organ failure in post infectious sequelae	scientific article published on January 1999

Statements

scholarly article

1 reference

Europe PubMed Central

16 August 2017

Multi-drug strategies are necessary to inhibit the synergistic mechanism causing tissue damage and organ failure in post infectious sequelae (English)

1 reference

Europe PubMed Central

16 August 2017

author name string

Ginsburg I

1

1 reference

Europe PubMed Central

16 August 2017

publication date

1 January 1999

1 reference

Europe PubMed Central

16 August 2017

Inflammopharmacology

1 reference

Europe PubMed Central

16 August 2017

7

1 reference

Europe PubMed Central

16 August 2017

3

1 reference

Europe PubMed Central

16 August 2017

207-217

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Europe PubMed Central

16 August 2017

https://scigraph.springernature.com/pub.10.1007/s10787-999-0004-1

0 references

The horror autotoxicus and multiple-organ failure

1 reference

https://api.crossref.org/works/10.1007%2FS10787-999-0004-1

21 January 2018

Protection by D-amino acids against growth inhibition and lysis caused by beta-lactam antibiotics

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https://api.crossref.org/works/10.1007%2FS10787-999-0004-1

21 January 2018

Tyrphostin AG 556 improves survival and reduces multiorgan failure in canine Escherichia coli peritonitis.

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https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Streptococcal toxic-shock syndrome: spectrum of disease, pathogenesis, and new concepts in treatment

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Synergistic cytolysis mediated by hydrogen peroxide combined with peptide defensins

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Potential strategies for inflammatory mediator manipulation: retrospect and prospect

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Could synergistic interactions among reactive oxygen species, proteinases, membrane-perforating enzymes, hydrolases, microbial hemolysins and cytokines be the main cause of tissue damage in infectious and inflammatory conditions?

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Treating patients with severe sepsis

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Mechanisms of cell and tissue injury induced by group A streptococci: relation to poststreptococcal sequelae

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Antiendotoxin strategies for the prevention and treatment of septic shock. New approaches and future directions

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Degradation of endothelial cell matrix heparan sulfate proteoglycan by elastase and the myeloperoxidase-H2O2-chloride system.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Direct effects of neutrophil oxidants on elastase-induced extracellular matrix proteolysis

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Gamma globulin, Evan's blue, aprotinin A PLA2 inhibitor, tetracycline and antioxidants protect epithelial cells against damage induced by synergism among streptococcal hemolysins, oxidants and proteinases: relation to the prevention of post-streptoc

1 reference

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12 December 2020

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The significance of endotoxin release in experimental and clinical sepsis in surgical patients--evidence for antibiotic-induced endotoxin release?

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Inhibition of the bacteriolytic effect of beta-lactam-antibiotics on Staphylococcus aureus by the polyanionic drugs suramin and Evans Blue.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Clinical trials in sepsis: where do we stand?

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Cell damage in inflammatory and infectious sites might involve a coordinated "cross-talk" among oxidants, microbial haemolysins and ampiphiles, cationic proteins, phospholipases, fatty acids, proteinases and cytokines (an overview).

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Anti-inflammatory therapies to treat sepsis and septic shock: a reassessment

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12 December 2020

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Neutrophil-mediated solubilization of the subendothelial matrix: oxidative and nonoxidative mechanisms of proteolysis used by normal and chronic granulomatous disease phagocytes

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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PADMA-28, a traditional tibetan herbal preparation inhibits the respiratory burst in human neutrophils, the killing of epithelial cells by mixtures of oxidants and pro-inflammatory agonists and peroxidation of lipids

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Therapeutic immunomodulatory approaches for the control of systemic inflammatory response syndrome and the prevention of sepsis

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Endothelial cell xanthine oxidase-derived toxic oxygen metabolites contribute to acute lung injury from neutrophil elastase

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Suppression of penicillin-induced bacteriolysis of staphylococci by some anticoagulants

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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The role of lysosomal factors of leukocytes in the biodegradation and storage of microbial constituents in infectious granulomas

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Organ-specific therapy in critical illness: interfacing molecular mechanisms with physiological interventions

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Antibiotic-induced release of bacterial cell wall components in the pathogenesis of sepsis and septic shock: a review

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Can hemolytic streptococci be considered "forefathers" of modern phagocytes? Both cell types freely migrate in tissues and destroy host cells by a "synergistic cross-talk" among their secreted agonists.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Anti-inflammatory therapies: application of molecular biology techniques in intensive care medicine

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Current status of antioxidant therapy

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https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Study of human epithelial cell detachment and damage: effects of proteases and oxidants

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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The biochemistry of bacteriolysis: paradoxes, facts and myths

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https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Interferon-gamma therapy for infectious complications of injury: a called third strike?

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Applications of molecular biology and biotechnology: antibody therapy of sepsis

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Role of cytokines in sepsis.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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The inflammatory basis of trauma/shock-associated multiple organ failure.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Inflammatory mediators, cytokines, and adhesion molecules in pulmonary inflammation and injury.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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The synergistic effect of elastase and hydrogen peroxide on vascular endothelial cell injury is due to the production of hydroxylradical in the endothelial cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Immunotherapy for sepsis

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Antibiotics, cytokines, and endotoxin: a complex and evolving relationship in gram-negative sepsis

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Cationic polyelectrolytes: a new look at their possible roles as opsonins, as stimulators of respiratory burst in leukocytes, in bacteriolysis, and as modulators of immune-complex diseases (a review hypothesis).

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Can we learn from the pathogenetic strategies of group A hemolytic streptococci how tissues are injured and organs fail in post-infectious and inflammatory sequelae?

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Effects of mucopolysaccharides on penicillin-induced lysis of Staphylococcus aureus

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Issues in the adjunct therapy of severe sepsis

1 reference

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12 December 2020

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H2O2 renders cells accessible to lysis by exogenous phospholipase A2: a novel mechanism for cell damage in inflammatory processes

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Tissue destruction by neutrophils

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Protection against septic shock and suppression of tumor necrosis factor alpha and nitric oxide production by dexanabinol (HU-211), a nonpsychotropic cannabinoid.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Sepsis research: we must change course

1 reference

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12 December 2020

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Systemic inflammatory response syndrome (SIRS), multiple organ dysfunction syndrome (MODS), multiple organ failure (MOF): are we winning the battle?

1 reference

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12 December 2020

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O2 metabolites and neutrophil elastase synergistically cause edematous injury in isolated rat lungs.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Pharmacologic antidotes in critical care medicine: a practical guide for drug administration

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

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Bacteriolysis is inhibited by hydrogen peroxide and by proteases

1 reference

https://pubmed.ncbi.nlm.nih.gov/17638092

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1007/S10787-999-0004-1

1 reference

Europe PubMed Central

16 August 2017

1 reference

Europe PubMed Central

16 August 2017

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