Transcription-independent turnover of I kappa B alpha during monocyte adherence: implications for a translational component regulating I kappa B alpha/MAD-3 mRNA levels (Q36550113)

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scientific article published on March 1995

Language	Label	Description	Also known as
English	Transcription-independent turnover of I kappa B alpha during monocyte adherence: implications for a translational component regulating I kappa B alpha/MAD-3 mRNA levels	scientific article published on March 1995

Statements

scholarly article

1 reference

Europe PubMed Central

14 August 2017

Transcription-independent turnover of I kappa B alpha during monocyte adherence: implications for a translational component regulating I kappa B alpha/MAD-3 mRNA levels (English)

1 reference

Europe PubMed Central

14 August 2017

author name string

A K Lofquist

1

1 reference

Europe PubMed Central

14 August 2017

K Mondal

2

1 reference

Europe PubMed Central

14 August 2017

J S Morris

3

1 reference

Europe PubMed Central

14 August 2017

J S Haskill

4

1 reference

Europe PubMed Central

14 August 2017

language of work or name

0 references

publication date

1 March 1995

1 reference

Europe PubMed Central

14 August 2017

Molecular and Cellular Biology

1 reference

Europe PubMed Central

14 August 2017

15

1 reference

Europe PubMed Central

14 August 2017

3

1 reference

Europe PubMed Central

14 August 2017

1737-1746

1 reference

Europe PubMed Central

14 August 2017

Lipopolysaccharide induces phosphorylation of MAD3 and activation of c-Rel and related NF-kappa B proteins in human monocytic THP-1 cells.

1 reference

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Tumor necrosis factor and interleukin-1 lead to phosphorylation and loss of I kappa B alpha: a mechanism for NF-kappa B activation

1 reference

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Endothelial cell gene expression in response to injury

1 reference

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Mutual regulation of the transcriptional activator NF-kappa B and its inhibitor, I kappa B-alpha

1 reference

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Regulation of eukaryotic gene expression by transcriptional attenuation

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Selective destabilization of short-lived mRNAs with the granulocyte-macrophage colony-stimulating factor AU-rich 3' noncoding region is mediated by a cotranslational mechanism

1 reference

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Signal transduction from the extracellular matrix

1 reference

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Rapid proteolysis of I kappa B-alpha is necessary for activation of transcription factor NF-kappa B

1 reference

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Cytokine-inducible expression in endothelial cells of an I kappa B alpha-like gene is regulated by NF kappa B.

1 reference

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Cytokine signal transduction

1 reference

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Autoregulation of I kappa B alpha activity

1 reference

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Promoter analysis of the gene encoding the I kappa B-alpha/MAD3 inhibitor of NF-kappa B: positive regulation by members of the rel/NF-kappa B family

1 reference

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Proteolytic degradation of MAD3 (I kappa B alpha) and enhanced processing of the NF-kappa B precursor p105 are obligatory steps in the activation of NF-kappa B.

1 reference

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In vivo control of NF-kappa B activation by I kappa B alpha.

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The I kappa B proteins: members of a multifunctional family

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NF-kappa B controls expression of inhibitor I kappa B alpha: evidence for an inducible autoregulatory pathway

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Messenger RNA degradation in eukaryotes

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Quantitation of mRNA by the polymerase chain reaction

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Monocyte adherence results in selective induction of novel genes sharing homology with mediators of inflammation and tissue repair

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Identifiers

10.1128/MCB.15.3.1737

1 reference

Europe PubMed Central

14 August 2017

1 reference

Europe PubMed Central

14 August 2017

1 reference

Europe PubMed Central

14 August 2017

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