Modulating processes within the central nervous system is central to therapeutic control of multiple sclerosis (Q36299405)

13 August 2017

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title

Modulating processes within the central nervous system is central to therapeutic control of multiple sclerosis (English)

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13 August 2017

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1

Tjalf Ziemssen

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13 August 2017

publication date

1 November 2005

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13 August 2017

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13 August 2017

volume

252 Suppl 5

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13 August 2017

page(s)

v38-45

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https://scigraph.springernature.com/pub.10.1007/s00415-005-5007-2

13 August 2017

exact match

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cites work

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Insulin-like growth factor-1 fails to enhance central nervous system myelin repair during autoimmune demyelination

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Mechanisms of action of glatiramer acetate in multiple sclerosis

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Tumor necrosis factor blockade in actively induced experimental autoimmune encephalomyelitis prevents clinical disease despite activated T cell infiltration to the central nervous system

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Cutting edge: clonally restricted production of the neurotrophins brain-derived neurotrophic factor and neurotrophin-3 mRNA by human immune cells and Th1/Th2-polarized expression of their receptors.

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BDNF and gp145trkB in multiple sclerosis brain lesions: neuroprotective interactions between immune and neuronal cells?

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The neuregulin, glial growth factor 2, diminishes autoimmune demyelination and enhances remyelination in a chronic relapsing model for multiple sclerosis

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Human nerve growth factor protects common marmosets against autoimmune encephalomyelitis by switching the balance of T helper cell type 1 and 2 cytokines within the central nervous system

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Glatiramer acetate-specific T-helper 1- and 2-type cell lines produce BDNF: implications for multiple sclerosis therapy. Brain-derived neurotrophic factor

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Intracortical multiple sclerosis lesions are not associated with increased lymphocyte infiltration

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LIF receptor signaling limits immune-mediated demyelination by enhancing oligodendrocyte survival

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Activated human T cells, B cells, and monocytes produce brain-derived neurotrophic factor in vitro and in inflammatory brain lesions: a neuroprotective role of inflammation?

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Neuroprotection and glatiramer acetate: the possible role in the treatment of multiple sclerosis.

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Adenoviral gene transfer of ciliary neurotrophic factor and brain-derived neurotrophic factor leads to long-term survival of axotomized motor neurons

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Axonal damage in multiple sclerosis: a complex issue in a complex disease

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Unimpaired autoreactive T-cell traffic within the central nervous system during tumor necrosis factor receptor-mediated inhibition of experimental autoimmune encephalomyelitis

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Minocycline reduces gadolinium-enhancing magnetic resonance imaging lesions in multiple sclerosis.

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Multiple sclerosis: a two-stage disease

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Minocycline, a tetracycline derivative, is neuroprotective against excitotoxicity by inhibiting activation and proliferation of microglia.

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10.1007/S00415-005-5007-2

Identifiers

DOI

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13 August 2017

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