The MMS22L-TONSL heterodimer directly promotes RAD51-dependent recombination upon replication stress. (Q36178409)

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26 March 2020

title

The MMS22L-TONSL heterodimer directly promotes RAD51-dependent recombination upon replication stress (English)

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26 March 2020

1

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26 March 2020

Lucie Mlejnkova

2

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26 March 2020

Alexey Smirnov

6

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26 March 2020

Matthias Peter

12

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26 March 2020

Petr Cejka

13

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26 March 2020

author name string

Karun Mutreja

3

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26 March 2020

Lepakshi Ranjha

4

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26 March 2020

Diana Stafa

5

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26 March 2020

Mia Ml Brodersen

7

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26 March 2020

Ralph Zellweger

8

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26 March 2020

Andreas Sturzenegger

9

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26 March 2020

Pavel Janscak

10

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26 March 2020

Massimo Lopes

11

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26 March 2020

language of work or name

English

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publication date

26 October 2016

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26 March 2020

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26 March 2020

volume

35

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26 March 2020

issue

23

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26 March 2020

page(s)

2584-2601

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describes a project that uses

26 March 2020

ImageJ

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11 June 2022

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based on heuristic

inferred from PubMed Central ID database lookup

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Germline mutation in the RAD51B gene confers predisposition to breast cancer

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FBH1 helicase disrupts RAD51 filaments in vitro and modulates homologous recombination in mammalian cells

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A variant of the breast cancer type 2 susceptibility protein (BRC) repeat is essential for the RECQL5 helicase to interact with RAD51 recombinase for genome stabilization

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RNAi-based screening identifies the Mms22L-Nfkbil2 complex as a novel regulator of DNA replication in human cells

2 July 2018

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A genome-wide camptothecin sensitivity screen identifies a mammalian MMS22L-NFKBIL2 complex required for genomic stability

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Identification of the MMS22L-TONSL complex that promotes homologous recombination

2 July 2018

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The MMS22L-TONSL complex mediates recovery from replication stress and homologous recombination

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2 July 2018

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2 July 2018

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PubMed

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12 December 2020

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Identifiers

DOI

10.15252/EMBJ.201593132

1 reference

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26 March 2020

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24 November 2022

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1 reference

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26 March 2020

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