Cerebellar dysfunction in multiple sclerosis: evidence for an acquired channelopathy (Q36015265)

11 August 2017

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title

Cerebellar dysfunction in multiple sclerosis: evidence for an acquired channelopathy (English)

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11 August 2017

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Stephen G Waxman

1

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11 August 2017

publication date

1 January 2005

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Progress in Brain Research

11 August 2017

published in

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11 August 2017

volume

148

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11 August 2017

page(s)

353-365

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A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons

11 August 2017

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Insertion of a SNS-specific tetrapeptide in S3-S4 linker of D4 accelerates recovery from inactivation of skeletal muscle voltage-gated Na channel mu1 in HEK293 cells

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Expression of Nav1.8 sodium channels perturbs the firing patterns of cerebellar Purkinje cells.

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Downregulation of Na+ channel mRNA in olfactory bulb tufted cells following deafferentation

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7 January 2021

Experimental and modeling study of Na+ current heterogeneity in rat nodose neurons and its impact on neuronal discharge.

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Subthreshold sodium current from rapidly inactivating sodium channels drives spontaneous firing of tuberomammillary neurons

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7 January 2021

Molecular and functional remodeling of electrogenic membrane of hypothalamic neurons in response to changes in their input

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7 January 2021

The neuron as a dynamic electrogenic machine: modulation of sodium-channel expression as a basis for functional plasticity in neurons

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7 January 2021

Transcriptional channelopathies: an emerging class of disorders

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7 January 2021

Ion channels and neuronal dysfunction in multiple sclerosis

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7 January 2021

Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy

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7 January 2021

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Identifiers

DOI

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11 August 2017

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