AbetaPP-overexpression and proteasome inhibition increase alphaB-crystallin in cultured human muscle: relevance to inclusion-body myositis. (Q35990823)

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21 December 2019

title

AbetaPP-overexpression and proteasome inhibition increase alphaB-crystallin in cultured human muscle: relevance to inclusion-body myositis (English)

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21 December 2019

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1

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21 December 2019

author name string

W King Engel

2

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21 December 2019

Janis McFerrin

3

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21 December 2019

Orlando Paciello

4

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21 December 2019

Valerie Askanas

5

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21 December 2019

publication date

23 October 2006

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21 December 2019

published in

Neuromuscular Disorders

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21 December 2019

volume

16

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21 December 2019

issue

12

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21 December 2019

page(s)

839-844

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Inclusion-body myositis: a myodegenerative conformational disorder associated with Abeta, protein misfolding, and proteasome inhibition

21 December 2019

cites work

1 reference

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Inflammatory, immune, and viral aspects of inclusion-body myositis

2 October 2017

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Inclusion-body myositis: clinical, diagnostic, and pathologic aspects

2 October 2017

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2 October 2017

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AlphaB-crystallin, a small heat-shock protein, prevents the amyloid fibril growth of an amyloid beta-peptide and beta2-microglobulin

2 October 2017

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Proteasome inhibition and aggresome formation in sporadic inclusion-body myositis and in amyloid-beta precursor protein-overexpressing cultured human muscle fibers

2 October 2017

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2 October 2017

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Endoplasmic reticulum stress and unfolded protein response in inclusion body myositis muscle.

2 October 2017

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Inclusion body myositis-like phenotype induced by transgenic overexpression of beta APP in skeletal muscle

2 October 2017

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Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

2 October 2017

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Inhibition of proteasomes induces accumulation, phosphorylation, and recruitment of HSP27 and alphaB-crystallin to aggresomes

2 October 2017

1 reference

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Epoxomicin, a potent and selective proteasome inhibitor, exhibits in vivo antiinflammatory activity

2 October 2017

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Alpha-crystallin as a molecular chaperone

2 October 2017

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Amyloid-beta deposition in skeletal muscle of transgenic mice: possible model of inclusion body myopathy

2 October 2017

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Transgenic mice over-expressing the C-99 fragment of betaPP with an alpha-secretase site mutation develop a myopathy similar to human inclusion body myositis

2 October 2017

1 reference

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A missense mutation in the alphaB-crystallin chaperone gene causes a desmin-related myopathy

2 October 2017

1 reference

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Transfer of beta-amyloid precursor protein gene using adenovirus vector causes mitochondrial abnormalities in cultured normal human muscle

2 October 2017

1 reference

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Myostatin is increased and complexes with amyloid-beta within sporadic inclusion-body myositis muscle fibers

2 October 2017

1 reference

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Transthyretin Val122Ile, accumulated Abeta, and inclusion-body myositis aspects in cultured muscle

11 July 2018

1 reference

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Beta APP gene transfer into cultured human muscle induces inclusion-body myositis aspects

11 July 2018

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Transglutaminase catalyzes differential crosslinking of small heat shock proteins and amyloid-beta

11 July 2018

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Targeting the neurotoxic species in Alzheimer's disease: inhibitors of Abeta oligomerization

23 September 2018

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Cystatin C colocalizes with amyloid-beta and coimmunoprecipitates with amyloid-beta precursor protein in sporadic inclusion-body myositis muscles.

23 September 2018

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Oligomeric and fibrillar species of amyloid-beta peptides differentially affect neuronal viability.

23 September 2018

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AlphaB-crystallin immunolocalization yields new insights into inclusion body myositis.

23 September 2018

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The molecular chaperone alphaB-crystallin enhances amyloid beta neurotoxicity.

23 September 2018

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Impaired innervation of cultured human muscle overexpressing betaAPP experimentally and genetically: relevance to inclusion-body myopathies

23 September 2018

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Biologically stressed muscle fibers in sporadic IBM: a clue for the enigmatic etiology?

23 September 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/17056255

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

ADDLs & protofibrils--the missing links?

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/17056255

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1016/J.NMD.2006.08.009

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17056255%20AND%20SRC:MED&resulttype=core&format=json

21 December 2019

1 reference

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21 December 2019

PubMed publication ID

17056255

1 reference