The interplay between microRNAs and the neurotrophin receptor tropomyosin-related kinase C controls proliferation of human neuroblastoma cells. (Q35808837)

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scholarly article

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English	The interplay between microRNAs and the neurotrophin receptor tropomyosin-related kinase C controls proliferation of human neuroblastoma cells.	scholarly article

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scholarly article

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30 December 2019

The interplay between microRNAs and the neurotrophin receptor tropomyosin-related kinase C controls proliferation of human neuroblastoma cells (English)

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30 December 2019

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4

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30 December 2019

Elisabetta Ferretti

5

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30 December 2019

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Pietro Laneve

1

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30 December 2019

Lucia Di Marcotullio

2

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30 December 2019

Ubaldo Gioia

3

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30 December 2019

Alberto Gulino

6

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30 December 2019

Irene Bozzoni

7

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30 December 2019

Elisa Caffarelli

8

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30 December 2019

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1 May 2007

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30 December 2019

Proceedings of the National Academy of Sciences of the United States of America

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30 December 2019

104

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30 December 2019

7957-7962

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30 December 2019

19

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30 December 2019

Comparative sequence analysis reveals an intricate network among REST, CREB and miRNA in mediating neuronal gene expression

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A minicircuitry comprised of microRNA-223 and transcription factors NFI-A and C/EBPalpha regulates human granulopoiesis

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MicroRNAs acting in a double-negative feedback loop to control a neuronal cell fate decision

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MicroRNAs regulate brain morphogenesis in zebrafish

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Microarray analysis of microRNA expression in the developing mammalian brain

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Expression profiling of mammalian microRNAs uncovers a subset of brain-expressed microRNAs with possible roles in murine and human neuronal differentiation

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Targeted deletion of all isoforms of the trkC gene suggests the use of alternate receptors by its ligand neurotrophin-3 in neuronal development and implicates trkC in normal cardiogenesis

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Targeted expression of MYCN causes neuroblastoma in transgenic mice

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Expression of mRNA for the neurotrophin receptor trkC in neuroblastomas with favourable tumour stage and good prognosis

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Conditional expression of N-myc in human neuroblastoma cells increases expression of alpha-prothymosin and ornithine decarboxylase and accelerates progression into S-phase early after mitogenic stimulation of quiescent cells.

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Naturally occurring truncated trkB receptors have dominant inhibitory effects on brain-derived neurotrophic factor signaling

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Disruption of the neurotrophin-3 receptor gene trkC eliminates la muscle afferents and results in abnormal movements

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Decreased expression of N-myc precedes retinoic acid-induced morphological differentiation of human neuroblastoma.

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Biology of pediatric peripheral neuroectodermal tumors

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Association of multiple copies of the N-myc oncogene with rapid progression of neuroblastomas

1 reference

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12 December 2020

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Expression of TrkC in favorable human neuroblastomas

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https://pubmed.ncbi.nlm.nih.gov/17483472

12 December 2020

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Identifiers

10.1073/PNAS.0700071104

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30 December 2019

2007PNAS..104.7957L

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30 December 2019

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30 December 2019

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