The role of apoptotic volume decrease and ionic homeostasis in the activation and repression of apoptosis (Q35755609)

10 August 2017

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title

The role of apoptotic volume decrease and ionic homeostasis in the activation and repression of apoptosis (English)

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10 August 2017

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1

Carl D Bortner

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10 August 2017

2

John A Cidlowski

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PubMed ID

15107996

10 August 2017

language of work or name

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24 April 2004

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10 August 2017

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10 August 2017

448

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10 August 2017

313-318

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10 August 2017

3

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https://scigraph.springernature.com/pub.10.1007/s00424-004-1266-5

10 August 2017

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cites work

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Human monocyte interleukin-1beta posttranslational processing. Evidence of a volume-regulated response

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Uncoupling cell shrinkage from apoptosis reveals that Na+ influx is required for volume loss during programmed cell death

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A necessary role for cell shrinkage in apoptosis

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Physiological concentrations of K+ inhibit cytochrome c-dependent formation of the apoptosome

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Plasma membrane depolarization without repolarization is an early molecular event in anti-Fas-induced apoptosis.

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Absence of volume regulatory mechanisms contributes to the rapid activation of apoptosis in thymocytes

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Normotonic cell shrinkage because of disordered volume regulation is an early prerequisite to apoptosis

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A primary role for K+ and Na+ efflux in the activation of apoptosis

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Ions, cell volume, and apoptosis

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Activation of K+ channels induces apoptosis in vascular smooth muscle cells

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Intracellular K+ suppresses the activation of apoptosis in lymphocytes

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Inhibition of Na+/K(+)-ATPase may be one mechanism contributing to potassium efflux and cell shrinkage in CD95-induced apoptosis

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Receptor-mediated control of regulatory volume decrease (RVD) and apoptotic volume decrease (AVD).

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Changes in elemental content during apoptotic cell death studied by electron probe X-ray microanalysis

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Apoptotic volume decrease and the incredible shrinking cell

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Stimulation of Kv1.3 potassium channels by death receptors during apoptosis in Jurkat T lymphocytes

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Elevated extracellular [K+] inhibits death-receptor- and chemical-mediated apoptosis prior to caspase activation and cytochrome c release

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A histochemical study of hypertrophy and ischaemic injury of rat liver with special reference to changes in lysosomes

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Shrinkage necrosis: a distinct mode of cellular death.

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Intracellular adenosine triphosphate (ATP) concentration: a switch in the decision between apoptosis and necrosis

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10.1007/S00424-004-1266-5

Identifiers

DOI

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10 August 2017

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PubMed ID

15107996