Lithium ameliorates neurodegeneration, suppresses neuroinflammation, and improves behavioral performance in a mouse model of traumatic brain injury (Q35682342)

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scientific article published on November 2011

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English	Lithium ameliorates neurodegeneration, suppresses neuroinflammation, and improves behavioral performance in a mouse model of traumatic brain injury	scientific article published on November 2011

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scholarly article

1 reference

Europe PubMed Central

9 August 2017

Lithium ameliorates neurodegeneration, suppresses neuroinflammation, and improves behavioral performance in a mouse model of traumatic brain injury (English)

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Europe PubMed Central

9 August 2017

traumatic brain injury

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6

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author name string

Fengshan Yu

1

1 reference

Europe PubMed Central

9 August 2017

Zhifei Wang

2

1 reference

Europe PubMed Central

9 August 2017

Flaubert Tchantchou

3

1 reference

Europe PubMed Central

9 August 2017

Chi-Tso Chiu

4

1 reference

Europe PubMed Central

9 August 2017

Yumin Zhang

5

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Europe PubMed Central

9 August 2017

publication date

1 November 2011

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Europe PubMed Central

9 August 2017

Journal of Neurotrauma

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Europe PubMed Central

9 August 2017

29

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Europe PubMed Central

9 August 2017

2

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Europe PubMed Central

9 August 2017

362-374

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Europe PubMed Central

9 August 2017

describes a project that uses

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Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3261788/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

Neuroprotection for traumatic brain injury: translational challenges and emerging therapeutic strategies

1 reference

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Valproic acid attenuates blood-brain barrier disruption in a rat model of transient focal cerebral ischemia: the roles of HDAC and MMP-9 inhibition.

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Molecular actions and therapeutic potential of lithium in preclinical and clinical studies of CNS disorders

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Is glycogen synthase kinase-3 a central modulator in mood regulation?

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Valproate administered after traumatic brain injury provides neuroprotection and improves cognitive function in rats

1 reference

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Lentivirally mediated GSK-3β silencing in the hippocampal dentate gyrus induces antidepressant-like effects in stressed mice

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Blood-brain barrier breakdown as a therapeutic target in traumatic brain injury

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Activated protein C analog with reduced anticoagulant activity improves functional recovery and reduces bleeding risk following controlled cortical impact

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Novel insights into lithium's mechanism of action: neurotrophic and neuroprotective effects

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Innate and adaptive immune responses regulated by glycogen synthase kinase-3 (GSK3).

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Neutralization of interleukin-1beta modifies the inflammatory response and improves histological and cognitive outcome following traumatic brain injury in mice.

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Alterations in blood-brain barrier permeability to large and small molecules and leukocyte accumulation after traumatic brain injury: effects of post-traumatic hypothermia

1 reference

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Combination therapies for traumatic brain injury: prospective considerations

1 reference

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Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury.

1 reference

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Glycogen synthase kinase-3 regulates microglial migration, inflammation, and inflammation-induced neurotoxicity

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Genetic disruption of cyclooxygenase-2 does not improve histological or behavioral outcome after traumatic brain injury in mice

1 reference

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Moderate and severe traumatic brain injury in adults.

1 reference

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Selective death of newborn neurons in hippocampal dentate gyrus following moderate experimental traumatic brain injury

1 reference

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Evolution of post-traumatic neurodegeneration after controlled cortical impact traumatic brain injury in mice and rats as assessed by the de Olmos silver and fluorojade staining methods

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Induction of mmp-9 expression and endothelial injury by oxidative stress after spinal cord injury

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MMP-9-positive neutrophil infiltration is associated to blood-brain barrier breakdown and basal lamina type IV collagen degradation during hemorrhagic transformation after human ischemic stroke.

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Selective loss of brain-derived neurotrophic factor in the dentate gyrus attenuates antidepressant efficacy

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Progressive damage after brain and spinal cord injury: pathomechanisms and treatment strategies

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In search of the Holy Grail for the treatment of neurodegenerative disorders: has a simple cation been overlooked?

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GSK-3 is a viable potential target for therapeutic intervention in bipolar disorder

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Differential roles of glycogen synthase kinase-3 isoforms in the regulation of transcriptional activation

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Lithium-mediated phosphorylation of glycogen synthase kinase-3beta involves PI3 kinase-dependent activation of protein kinase C-alpha

1 reference

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Motor and cognitive function evaluation following experimental traumatic brain injury

1 reference

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Lithium and GSK-3: one inhibitor, two inhibitory actions, multiple outcomes

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Postinsult treatment with lithium reduces brain damage and facilitates neurological recovery in a rat ischemia/reperfusion model

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Cyclooxygenase-2 contributes to functional hyperemia in whisker-barrel cortex.

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1 reference

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Effects of genetic deficiency of cyclooxygenase-1 or cyclooxygenase-2 on functional and histological outcomes following traumatic brain injury in mice

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Depression-like and anxiety-like behavioural aftermaths of impact accelerated traumatic brain injury in rats: a model of comorbid depression and anxiety?

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Inhibition of GSK-3 reduces infarct volume and improves neurobehavioral functions

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The mood stabilizers lithium and valproate selectively activate the promoter IV of brain-derived neurotrophic factor in neurons.

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Role of glycogen synthase kinase-3beta in early depressive behavior induced by mild traumatic brain injury.

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Regulation and function of glycogen synthase kinase-3 isoforms in neuronal survival.

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Effects of the selective cyclooxygenase-2 inhibitor rofecoxib on cell death following traumatic brain injury in the rat.

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Lithium activates the serine/threonine kinase Akt-1 and suppresses glutamate-induced inhibition of Akt-1 activity in neurons

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12 December 2020

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Identifiers

10.1089/NEU.2011.1942

1 reference

Europe PubMed Central

9 August 2017

1 reference

Europe PubMed Central

9 August 2017

1 reference

Europe PubMed Central

9 August 2017

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