Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of Alda-1 (Q35510238)

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24 February 2020

title

Aldehydic load and aldehyde dehydrogenase 2 profile during the progression of post-myocardial infarction cardiomyopathy: benefits of Alda-1 (English)

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24 February 2020

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5

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24 February 2020

Julio Cesar Ferreira

9

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7

Alicia J Kowaltowski

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24 February 2020

author name string

Katia M S Gomes

1

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24 February 2020

Luiz R G Bechara

2

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24 February 2020

Vanessa M Lima

3

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24 February 2020

Márcio A C Ribeiro

4

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24 February 2020

Paulo M Dourado

6

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24 February 2020

Daria Mochly-Rosen

8

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publication date

23 October 2014

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International Journal of Cardiology

24 February 2020

published in

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volume

179

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page(s)

129-138

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Aldehyde dehydrogenase 2 activation in heart failure restores mitochondrial function and improves ventricular function and remodelling.

24 February 2020

cites work

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Acute inhibition of excessive mitochondrial fission after myocardial infarction prevents long-term cardiac dysfunction

27 July 2018

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Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) phosphorylation by protein kinase Cδ (PKCδ) inhibits mitochondria elimination by lysosomal-like structures following ischemia and reoxygenation-induced injury

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Mitochondria as a therapeutic target in heart failure

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Protein quality control disruption by PKCβII in heart failure; rescue by the selective PKCβII inhibitor, βIIV5-3

27 July 2018

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27 July 2018

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ALDH2 activator inhibits increased myocardial infarction injury by nitroglycerin tolerance

27 July 2018

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Nitroglycerin use in myocardial infarction patients

27 July 2018

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Pharmacological inhibition of βIIPKC is cardioprotective in late-stage hypertrophy

27 July 2018

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PKCβII inhibition attenuates myocardial infarction induced heart failure and is associated with a reduction of fibrosis and pro-inflammatory responses

27 July 2018

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Aldehyde dehydrogenase 2 knockout accentuates ethanol-induced cardiac depression: role of protein phosphatases

27 July 2018

1 reference

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Activation of aldehyde dehydrogenase 2 (ALDH2) confers cardioprotection in protein kinase C epsilon (PKCvarepsilon) knockout mice

27 July 2018

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Activation of aldehyde dehydrogenase-2 reduces ischemic damage to the heart

27 July 2018

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Reactions of 4-hydroxynonenal with proteins and cellular targets

27 July 2018

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Direct evidence that oxygen-derived free radicals contribute to postischemic myocardial dysfunction in the intact dog.

27 July 2018

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Targeting aldehyde dehydrogenase 2: new therapeutic opportunities

27 July 2018

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Impact of exercise training on redox signaling in cardiovascular diseases.

26 September 2018

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Exercise training restores cardiac protein quality control in heart failure

26 September 2018

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Regulation of mitochondrial processes: a target for heart failure

26 September 2018

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Aerobic exercise training improves skeletal muscle function and Ca2+ handling-related protein expression in sympathetic hyperactivity-induced heart failure.

26 September 2018

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Aldehyde dehydrogenase 2 ameliorates acute cardiac toxicity of ethanol: role of protein phosphatase and forkhead transcription factor

26 September 2018

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Metabolic remodeling induced by mitochondrial aldehyde stress stimulates tolerance to oxidative stress in the heart.

26 September 2018

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Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation

26 September 2018

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Pharmacological and physiological stimuli do not promote Ca(2+)-sensitive K+ channel activity in isolated heart mitochondria.

26 September 2018

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Relationship between oxidative stress and systolic dysfunction in patients with hypertrophic cardiomyopathy

26 September 2018

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Carvedilol decreases elevated oxidative stress in human failing myocardium

26 September 2018

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26 September 2018

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12 December 2020

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Unsaturated aldehydes including 4-OH-nonenal are elevated in patients with congestive heart failure

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Identifiers

DOI

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24 February 2020

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24 February 2020

PubMed publication ID

25464432

1 reference

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