Transforming growth factor-beta1 (TGF-beta1) utilizes distinct pathways for the transcriptional activation of microRNA 143/145 in human coronary artery smooth muscle cells (Q35314346)

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English	Transforming growth factor-beta1 (TGF-beta1) utilizes distinct pathways for the transcriptional activation of microRNA 143/145 in human coronary artery smooth muscle cells	scientific article

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scholarly article

1 reference

Europe PubMed Central

7 August 2017

Transforming growth factor-beta1 (TGF-beta1) utilizes distinct pathways for the transcriptional activation of microRNA 143/145 in human coronary artery smooth muscle cells (English)

1 reference

Europe PubMed Central

7 August 2017

1 reference

based on heuristic

inferred from title

author name string

Xiaochun Long

1

1 reference

Europe PubMed Central

7 August 2017

Joseph M Miano

2

1 reference

Europe PubMed Central

7 August 2017

language of work or name

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publication date

28 June 2011

1 reference

Europe PubMed Central

7 August 2017

Journal of Biological Chemistry

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Europe PubMed Central

7 August 2017

286

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Europe PubMed Central

7 August 2017

30119-30129

1 reference

Europe PubMed Central

7 August 2017

34

1 reference

Europe PubMed Central

7 August 2017

down-regulation of Kruppel-like factor-4 (KLF4) by microRNA-143/145 is critical for modulation of vascular smooth muscle cell phenotype by transforming growth factor-beta and bone morphogenetic protein 4

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

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Serum response factor-dependent MicroRNAs regulate gastrointestinal smooth muscle cell phenotypes

1 reference

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Pervasive roles of microRNAs in cardiovascular biology

1 reference

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NADPH oxidase 4 mediates TGF-β-induced smooth muscle α-actin via p38MAPK and serum response factor

1 reference

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27 July 2018

MicroRNA-1 regulates smooth muscle cell differentiation by repressing Kruppel-like factor 4

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

27 July 2018

Expression of micro-RNA-145 is regulated by a highly conserved genomic sequence 3' to the pre-miR.

1 reference

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Notch and transforming growth factor-beta (TGFbeta) signaling pathways cooperatively regulate vascular smooth muscle cell differentiation

1 reference

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MicroRNAs add a new dimension to cardiovascular disease

1 reference

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miR-10a contributes to retinoid acid-induced smooth muscle cell differentiation

1 reference

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Concordant regulation of translation and mRNA abundance for hundreds of targets of a human microRNA

1 reference

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MicroRNAs in cancer: small molecules with a huge impact

1 reference

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The knockout of miR-143 and -145 alters smooth muscle cell maintenance and vascular homeostasis in mice: correlates with human disease

1 reference

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The smooth muscle cell-restricted KCNMB1 ion channel subunit is a direct transcriptional target of serum response factor and myocardin.

1 reference

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MicroRNAs miR-143 and miR-145 modulate cytoskeletal dynamics and responsiveness of smooth muscle cells to injury

1 reference

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Acquisition of the contractile phenotype by murine arterial smooth muscle cells depends on the Mir143/145 gene cluster

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

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TGF-beta activates Akt kinase through a microRNA-dependent amplifying circuit targeting PTEN

1 reference

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MicroRNA-145, a novel smooth muscle cell phenotypic marker and modulator, controls vascular neointimal lesion formation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

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MicroRNA-145 regulates OCT4, SOX2, and KLF4 and represses pluripotency in human embryonic stem cells

1 reference

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miR-145 inhibits breast cancer cell growth through RTKN.

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Estradiol downregulates miR-21 expression and increases miR-21 target gene expression in MCF-7 breast cancer cells

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Airway smooth muscle in asthma: phenotype plasticity and function

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SRF and myocardin regulate LRP-mediated amyloid-beta clearance in brain vascular cells

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Induction of microRNA-221 by platelet-derived growth factor signaling is critical for modulation of vascular smooth muscle phenotype

1 reference

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MicroRNA-155 is regulated by the transforming growth factor beta/Smad pathway and contributes to epithelial cell plasticity by targeting RhoA

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

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miR-19, miR-101 and miR-130 co-regulate ATXN1 levels to potentially modulate SCA1 pathogenesis

1 reference

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SMAD proteins control DROSHA-mediated microRNA maturation

1 reference

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Transforming growth factor-beta-regulated miR-24 promotes skeletal muscle differentiation

1 reference

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Micro RNA 145 targets the insulin receptor substrate-1 and inhibits the growth of colon cancer cells

1 reference

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MicroRNA expression signature and antisense-mediated depletion reveal an essential role of MicroRNA in vascular neointimal lesion formation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

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Developmental basis of vascular smooth muscle diversity

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Myocardin enhances Smad3-mediated transforming growth factor-beta1 signaling in a CArG box-independent manner: Smad-binding element is an important cis element for SM22alpha transcription in vivo

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Synthetic retinoid Am80 suppresses smooth muscle phenotypic modulation and in-stent neointima formation by inhibiting KLF5.

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Transforming growth factor-beta1-induced expression of smooth muscle marker genes involves activation of PKN and p38 MAPK.

1 reference

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Serum response factor regulates a muscle-specific microRNA that targets Hand2 during cardiogenesis

1 reference

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AKAP12alpha, an atypical serum response factor-dependent target gene

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Molecular regulation of vascular smooth muscle cell differentiation in development and disease

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Transforming growth factor-beta-induced differentiation of smooth muscle from a neural crest stem cell line

1 reference

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Smad-dependent and Smad-independent pathways in TGF-beta family signalling

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Serum response factor: toggling between disparate programs of gene expression

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Myocardin is a master regulator of smooth muscle gene expression

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

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Myocardin is a key regulator of CArG-dependent transcription of multiple smooth muscle marker genes

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

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Myocardin is a critical serum response factor cofactor in the transcriptional program regulating smooth muscle cell differentiation.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3191051

27 July 2018

Transforming growth factor-beta induction of smooth muscle cell phenotpye requires transcriptional and post-transcriptional control of serum response factor.

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Phosphatidylinositol 3-kinase function is required for transforming growth factor beta-mediated epithelial to mesenchymal transition and cell migration.

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PDGF, TGF-beta, and heterotypic cell-cell interactions mediate endothelial cell-induced recruitment of 10T1/2 cells and their differentiation to a smooth muscle fate

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Modulation of actin dynamics during stress and physiological stimulation by a signaling pathway involving p38 MAP kinase and heat-shock protein 27.

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MicroRNA-26a is a novel regulator of vascular smooth muscle cell function

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Mammalian microRNAs predominantly act to decrease target mRNA levels

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miR-145 and miR-143 regulate smooth muscle cell fate and plasticity

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A necessary role of miR-221 and miR-222 in vascular smooth muscle cell proliferation and neointimal hyperplasia

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Myocardin is sufficient for a smooth muscle-like contractile phenotype

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Inactivation of myocardin and p16 during malignant transformation contributes to a differentiation defect.

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5' CArG degeneracy in smooth muscle alpha-actin is required for injury-induced gene suppression in vivo

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Transforming growth factor-beta1 signaling contributes to development of smooth muscle cells from embryonic stem cells.

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Smad3 mediates transforming growth factor-beta-induced alpha-smooth muscle actin expression

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Involvement of the p38 mitogen-activated protein kinase pathway in transforming growth factor-beta-induced gene expression

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https://pubmed.ncbi.nlm.nih.gov/21712382

12 December 2020

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Upregulation of intermediate-conductance Ca2+-activated K+ channel (IKCa1) mediates phenotypic modulation of coronary smooth muscle

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https://pubmed.ncbi.nlm.nih.gov/21712382

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MicroRNAs make big impression in disease after disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/21712382

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Central role for Rho in TGF-beta1-induced alpha-smooth muscle actin expression during epithelial-mesenchymal transition

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p38 MAP kinase modulates Smad-dependent changes in human prostate cell adhesion

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Identifiers

10.1074/JBC.M111.258814

1 reference

Europe PubMed Central

7 August 2017

1 reference

Europe PubMed Central

7 August 2017

1 reference

Europe PubMed Central

7 August 2017

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