Role of the aryl hydrocarbon receptor in cell cycle regulation. (Q35035489)

5 August 2017

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title

Role of the aryl hydrocarbon receptor in cell cycle regulation. (English)

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5 August 2017

Alvaro Puga

1

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5 August 2017

Jennifer Marlowe

2

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5 August 2017

Sonya Barnes

3

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5 August 2017

Ching-yi Chang

4

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5 August 2017

Andrew Maier

5

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5 August 2017

Zongqing Tan

6

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5 August 2017

J Kevin Kerzee

7

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5 August 2017

Xaoqing Chang

8

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5 August 2017

Matt Strobeck

9

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5 August 2017

Erik S Knudsen

10

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5 August 2017

language of work or name

English

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publication date

1 December 2002

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5 August 2017

7

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inferred from page(s)

published in

Toxicology

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5 August 2017

181-182

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5 August 2017

171-177

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Review of the interaction between TCDD and glucocorticoids in embryonic palate.

5 August 2017

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The mechanism of dioxin toxicity: relationship to risk assessment

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Partial demasculinization and feminization of sex behavior in male rats by in utero and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin is not associated with alterations in estrogen receptor binding or volumes of sexually differentiated

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Induction of highly proliferative phenotypes in cultured glomerular mesangial cells by benzo[a]pyrene alone or in combination with methoxamine

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Cloning of the Ah-receptor cDNA reveals a distinctive ligand-activated transcription factor

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Ligand-dependent interaction of the aryl hydrocarbon receptor with a novel immunophilin homolog in vivo

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Constitutive activation of the aromatic hydrocarbon receptor

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Dioxins: model chemicals for assessing receptor-mediated toxicity.

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cDNA cloning and structure of mouse putative Ah receptor

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Aryl-hydrocarbon receptor-deficient mice are resistant to 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced toxicity.

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Immune system impairment and hepatic fibrosis in mice lacking the dioxin-binding Ah receptor

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Cancer mortality in workers exposed to 2,3,7,8-tetrachlorodibenzo- p-dioxin

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Exposure to polychlorinated dioxins and furans (PCDD/F) and mortality in a cohort of workers from a herbicide-producing plant in Hamburg, Federal Republic of Germany.

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Regulation of gene expression and acceleration of differentiation in human keratinocytes by 2,3,7,8-tetrachlorodibenzo-p-dioxin

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A direct interaction between the aryl hydrocarbon receptor and retinoblastoma protein. Linking dioxin signaling to the cell cycle

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Inhibition of growth by 2,3,7,8-tetrachlorodibenzo-p-dioxin in 5L rat hepatoma cells is associated with the presence of Ah receptor

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Evolutionary conservation of the vertebrate Ah (dioxin) receptor: amplification and sequencing of the PAS domain of a teleost Ah receptor cDNA.

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The aryl hydrocarbon receptor complex

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pRB plays an essential role in cell cycle arrest induced by DNA damage

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TGF beta-induced growth inhibition in primary fibroblasts requires the retinoblastoma protein

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Altered cell cycle kinetics, gene expression, and G1 restriction point regulation in Rb-deficient fibroblasts

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Cloning of a Factor Required for Activity of the Ah (Dioxin) Receptor

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7 January 2021

Evidence for the induction of apoptosis in thymocytes by 2,3,7,8-tetrachlorodibenzo-p-dioxin in vivo

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Immunological effects of chlorinated dibenzo-p-dioxins

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7 January 2021

Response of murine epidermis to 2,3,7,8-tetrachlorodibenzo-p-dioxin: Interaction of the Ah and hr loci

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7 January 2021

Results of a two-year chronic toxicity and oncogenicity study of 2,3,7,8-tetrachlorodibenzo-p-dioxin in rats

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7 January 2021

SM22 alpha, a marker of adult smooth muscle, is expressed in multiple myogenic lineages during embryogenesis.

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Receptor mechanisms and dose-response models for the effects of dioxins

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7 January 2021

A novel cytoplasmic protein that interacts with the Ah receptor, contains tetratricopeptide repeat motifs, and augments the transcriptional response to 2,3,7,8-tetrachlorodibenzo-p-dioxin

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Cancer mortality among workers in chemical plant contaminated with dioxin

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2,3,7,8-Tetrachlorodibenzo-p-dioxin kills immature thymocytes by Ca2+-mediated endonuclease activation

1 reference

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7 January 2021

2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) kills glucocorticoid-sensitive thymocytes in vivo

1 reference

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7 January 2021

2,3,7,8-Tetrachlorodibenzo-p-dioxin induces hyperplasia in confluent cultures of human keratinocytes

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7 January 2021

Control of pRB phosphorylation

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7 January 2021

Quantitative analysis of enzyme-altered liver foci in rats initiated with diethylnitrosamine and promoted with 2,3,7,8-tetrachlorodibenzo-p-dioxin or 1,2,3,4,6,7,8-heptachlorodibenzo-p-dioxin

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Proposed role of drug-metabolizing enzymes: regulation of steady state levels of the ligands that effect growth, homeostasis, differentiation, and neuroendocrine functions

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Phenotypic analysis of spleen, thymus, and peripheral blood cells in aged C57B1/6 mice following long-term exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin.

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7 January 2021

Aromatic hydrocarbon receptor interaction with the retinoblastoma protein potentiates repression of E2F-dependent transcription and cell cycle arrest

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7 January 2021

The murine Cyp1a-1 gene negatively regulates its own transcription and that of other members of the aromatic hydrocarbon-responsive [Ah] gene battery.

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7 January 2021

Restoration of retinoblastoma mediated signaling to Cdk2 results in cell cycle arrest

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7 January 2021

Complementation of Ah receptor deficiency in hepatoma cells: negative feedback regulation and cell cycle control by the Ah receptor

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7 January 2021

Toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in vitro: H4IIEC3-derived 5L hepatoma cells as a model system

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7 January 2021

Growth stimulation of primary rat hepatocytes by 2,3,7,8-tetrachlorodibenzo-p-dioxin

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7 January 2021

10.1016/S0300-483X(02)00276-7

Identifiers

DOI

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5 August 2017

1 reference