Downregulation of OPA3 is responsible for transforming growth factor-β-induced mitochondrial elongation and F-actin rearrangement in retinal pigment epithelial ARPE-19 cells (Q34713262)

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English	Downregulation of OPA3 is responsible for transforming growth factor-β-induced mitochondrial elongation and F-actin rearrangement in retinal pigment epithelial ARPE-19 cells	scientific article

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scholarly article

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Europe PubMed Central

4 August 2017

Downregulation of OPA3 is responsible for transforming growth factor-β-induced mitochondrial elongation and F-actin rearrangement in retinal pigment epithelial ARPE-19 cells (English)

1 reference

Europe PubMed Central

4 August 2017

object named as

Jonghee Yoon

2

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author name string

Seung-Wook Ryu

1

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Europe PubMed Central

4 August 2017

Nambin Yim

3

1 reference

Europe PubMed Central

4 August 2017

Kyungsun Choi

4

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Europe PubMed Central

4 August 2017

Chulhee Choi

5

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Europe PubMed Central

4 August 2017

language of work or name

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publication date

3 May 2013

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Europe PubMed Central

4 August 2017

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Europe PubMed Central

4 August 2017

8

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Europe PubMed Central

4 August 2017

5

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Europe PubMed Central

4 August 2017

e63495

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Europe PubMed Central

4 August 2017

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Creative Commons Attribution 4.0 International

3 May 2013

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April 2022 Public Data File from Crossref

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Pirfenidone inhibits transforming growth factor-β1-induced fibrogenesis by blocking nuclear translocation of Smads in human retinal pigment epithelial cell line ARPE-19.

1 reference

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1 reference

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Mitochondrial remodeling following fission inhibition by 15d-PGJ2 involves molecular changes in mitochondrial fusion protein OPA1.

1 reference

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Optic atrophy 3 as a protein of the mitochondrial outer membrane induces mitochondrial fragmentation

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Loss of MARCH5 mitochondrial E3 ubiquitin ligase induces cellular senescence through dynamin-related protein 1 and mitofusin 1

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Mechanism of TGF-beta signaling to growth arrest, apoptosis, and epithelial-mesenchymal transition

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TRAF6 mediates Smad-independent activation of JNK and p38 by TGF-beta

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Mitochondrial fission and fusion mediators, hFis1 and OPA1, modulate cellular senescence

1 reference

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Biogenesis and dynamics of mitochondria during the cell cycle: significance of 3'UTRs

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Smad transcription factors

1 reference

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Emerging functions of mammalian mitochondrial fusion and fission

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Specificity and versatility in tgf-beta signaling through Smads

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Regulation of the polarity protein Par6 by TGFbeta receptors controls epithelial cell plasticity

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Epithelial-mesenchymal transitions in development and pathologies

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Transcriptional control by the TGF-beta/Smad signaling system

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Cell cycle dependent morphology changes and associated mitochondrial DNA redistribution in mitochondria of human cell lines.

1 reference

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Dynamics of mitochondria during the cell cycle.

1 reference

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Drp1 phosphorylation and mitochondrial regulation.

1 reference

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21 January 2018

Identifiers

10.1371/JOURNAL.PONE.0063495

1 reference

Europe PubMed Central

4 August 2017

2013PLoSO...863495R

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1 reference

Europe PubMed Central

4 August 2017

1 reference

Europe PubMed Central

4 August 2017

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