Hindbrain patterning revisited: timing and effects of retinoic acid signalling. (Q34464611)

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5 December 2019

review article

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title

Hindbrain patterning revisited: timing and effects of retinoic acid signalling (English)

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5 December 2019

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2

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5 December 2019

author name string

Begemann G

series ordinal

1

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5 December 2019

language of work or name

English

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publication date

1 November 2001

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5 December 2019

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5 December 2019

volume

23

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5 December 2019

issue

11

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5 December 2019

page(s)

981-986

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Retinoic acid and pattern formation in vertebrates

5 December 2019

cites work

1 reference

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Defects in embryonic hindbrain development and fetal resorption resulting from vitamin A deficiency in the rat are prevented by feeding pharmacological levels of all-trans-retinoic acid

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Hindbrain respecification in the retinoid-deficient quail.

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Molecular identification of a major retinoic-acid-synthesizing enzyme, a retinaldehyde-specific dehydrogenase

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Metabolic inactivation of retinoic acid by a novel P450 differentially expressed in developing mouse embryos

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Complementary domains of retinoic acid production and degradation in the early chick embryo

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The retinoic acid-inactivating enzyme CYP26 is essential for establishing an uneven distribution of retinoic acid along the anterio-posterior axis within the mouse embryo

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1 reference

The retinoic acid-metabolizing enzyme, CYP26A1, is essential for normal hindbrain patterning, vertebral identity, and development of posterior structures

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Increased XRALDH2 activity has a posteriorizing effect on the central nervous system of Xenopus embryos

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Reprogramming Hox expression in the vertebrate hindbrain: influence of paraxial mesoderm and rhombomere transposition.

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Initiation of rhombomeric Hoxb4 expression requires induction by somites and a retinoid pathway.

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Convergent inductive signals specify midbrain, hindbrain, and spinal cord identity in gastrula stage chick embryos.

21 January 2018

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A series of normal stages in the development of the chick embryo

21 January 2018

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Retinoids and related signals in early development of the vertebrate central nervous system.

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