NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFbeta1-induced fibroblast differentiation into myofibroblasts. (Q34415231)

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English	NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFbeta1-induced fibroblast differentiation into myofibroblasts.	scientific article

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9 February 2020

NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFbeta1-induced fibroblast differentiation into myofibroblasts (English)

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9 February 2020

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pulmonary fibrosis

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inferred from title

idiopathic pulmonary fibrosis

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Jorge Boczkowski

6

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9 February 2020

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Nadia Amara

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9 February 2020

Delphine Goven

2

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9 February 2020

Fabienne Prost

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9 February 2020

Rachel Muloway

4

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9 February 2020

Bruno Crestani

5

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9 February 2020

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1 August 2010

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9 February 2020

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9 February 2020

65

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9 February 2020

733-738

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9 February 2020

8

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9 February 2020

NADPH oxidase-4 mediates myofibroblast activation and fibrogenic responses to lung injury.

1 reference

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6 July 2018

Idiopathic pulmonary fibrosis: multiple causes and multiple mechanisms?

1 reference

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1 reference

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The myofibroblast: one function, multiple origins

1 reference

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1 reference

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Diesel exhaust particles induce matrix metalloprotease-1 in human lung epithelial cells via a NADP(H) oxidase/NOX4 redox-dependent mechanism.

1 reference

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Clinical review: idiopathic pulmonary fibrosis--past, present and future

1 reference

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6 July 2018

NAD(P)H oxidase 4 mediates transforming growth factor-beta1-induced differentiation of cardiac fibroblasts into myofibroblasts

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Reactive oxygen species production via NADPH oxidase mediates TGF-beta-induced cytoskeletal alterations in endothelial cells

1 reference

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Point mutations in the proline-rich region of p22phox are dominant inhibitors of Nox1- and Nox2-dependent reactive oxygen generation.

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Direct interaction of the novel Nox proteins with p22phox is required for the formation of a functionally active NADPH oxidase

1 reference

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6 July 2018

NOX enzymes and the biology of reactive oxygen

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Defect of hepatocyte growth factor secretion by fibroblasts in idiopathic pulmonary fibrosis

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TGF-beta1-mediated fibroblast-myofibroblast terminal differentiation-the role of Smad proteins

1 reference

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How cells read TGF-beta signals

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The fibronectin domain ED-A is crucial for myofibroblastic phenotype induction by transforming growth factor-beta1.

1 reference

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6 July 2018

The roles of the myofibroblast in idiopathic pulmonary fibrosis. Ultrastructural and immunohistochemical features of sites of active extracellular matrix synthesis

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The use of diphenylene iodonium and its analogues to investigate the role of the NADPH oxidase in the tumoricidal activity of macrophages in vitro

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NOX4 regulates ROS levels under normoxic and hypoxic conditions, triggers proliferation, and inhibits apoptosis in pulmonary artery adventitial fibroblasts

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Hypoxia-dependent regulation of nonphagocytic NADPH oxidase subunit NOX4 in the pulmonary vasculature.

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Transforming growth factor-beta1 induces Nox4 NAD(P)H oxidase and reactive oxygen species-dependent proliferation in human pulmonary artery smooth muscle cells

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12 December 2020

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In situ hybridization analysis of rat lung alpha 1(I) and alpha 2(I) collagen gene expression in pulmonary fibrosis induced by endotracheal bleomycin injection

1 reference

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12 December 2020

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TGF-beta 1, but not TGF-beta 2 or TGF-beta 3, is differentially present in epithelial cells of advanced pulmonary fibrosis: an immunohistochemical study

1 reference

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12 December 2020

based on heuristic

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10.1136/THX.2009.113456

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9 February 2020

1 reference

Europe PubMed Central

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9 February 2020

1 reference

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9 February 2020

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