Selectively frequent expression of CXCR5 enhances resistance to apoptosis in CD8(+)CD34(+) T cells from patients with T-cell-lineage acute lymphocytic leukemia (Q34373668)

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English	Selectively frequent expression of CXCR5 enhances resistance to apoptosis in CD8(+)CD34(+) T cells from patients with T-cell-lineage acute lymphocytic leukemia	scientific article

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1 August 2017

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Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine

Selectively frequent expression of CXCR5 enhances resistance to apoptosis in CD8(+)CD34(+) T cells from patients with T-cell-lineage acute lymphocytic leukemia (English)

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1 August 2017

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author name string

Zhang Qiuping

1

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1 August 2017

Xiong Jie

2

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1 August 2017

Jin Youxin

3

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1 August 2017

Wu Qun

4

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1 August 2017

Ju Wei

5

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1 August 2017

Liu Chun

6

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1 August 2017

Wang Jin

7

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1 August 2017

Liu Yan

8

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Hu Chunsong

9

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1 August 2017

Yang Mingzhen

10

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1 August 2017

Gao Qingping

11

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1 August 2017

Li Qun

12

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1 August 2017

Zhang Kejian

13

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1 August 2017

Sun Zhimin

14

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1 August 2017

Liu Junyan

15

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1 August 2017

Tan Jinquan

16

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1 August 2017

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1 January 2005

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24

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573-584

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1 August 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1208184

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CCR5 mediates Fas- and caspase-8 dependent apoptosis of both uninfected and HIV infected primary human CD4 T cells.

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A chemokine-driven positive feedback loop organizes lymphoid follicles

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The morphological classification of acute lymphoblastic leukaemia: concordance among observers and clinical correlations

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Proposals for the classification of chronic (mature) B and T lymphoid leukaemias. French-American-British (FAB) Cooperative Group

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CXCR4 and CD4 mediate a rapid CD95-independent cell death in CD4(+) T cells

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Follicular B helper T cells express CXC chemokine receptor 5, localize to B cell follicles, and support immunoglobulin production

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Structural basis of caspase-7 inhibition by XIAP

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A putative chemokine receptor, BLR1, directs B cell migration to defined lymphoid organs and specific anatomic compartments of the spleen

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A B-cell-homing chemokine made in lymphoid follicles activates Burkitt's lymphoma receptor-1

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A coordinated change in chemokine responsiveness guides plasma cell movements

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Induction of interleukin 10-producing, nonproliferating CD4(+) T cells with regulatory properties by repetitive stimulation with allogeneic immature human dendritic cells

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A CXC chemokine receptor, CXCR5/BLR1, is a novel and specific coreceptor for human immunodeficiency virus type 2.

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Livin, a novel inhibitor of apoptosis protein family member

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Involvement of BAFF and APRIL in the resistance to apoptosis of B-CLL through an autocrine pathway.

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Subspecialization of CXCR5+ T cells: B helper activity is focused in a germinal center-localized subset of CXCR5+ T cells

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KIAP, a novel member of the inhibitor of apoptosis protein family

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Biologically active APRIL is secreted following intracellular processing in the Golgi apparatus by furin convertase

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BLC expression in pancreatic islets causes B cell recruitment and lymphotoxin-dependent lymphoid neogenesis.

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Neisseria meningitidis porin PorB interacts with mitochondria and protects cells from apoptosis

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BCA-1 is highly expressed in Helicobacter pylori-induced mucosa-associated lymphoid tissue and gastric lymphoma

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The uncertain glory of APRIL.

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An exegesis of IAPs: salvation and surprises from BIR motifs

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The impact of CCR7 and CXCR5 on lymphoid organ development and systemic immunity

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Cooperating mechanisms of CXCR5 and CCR7 in development and organization of secondary lymphoid organs

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Chemokine requirements for B cell entry to lymph nodes and Peyer's patches.

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An endogenous hybrid mRNA encodes TWE-PRIL, a functional cell surface TWEAK-APRIL fusion protein

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Balanced responsiveness to chemoattractants from adjacent zones determines B-cell position

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A soluble form of B cell maturation antigen, a receptor for the tumor necrosis factor family member APRIL, inhibits tumor cell growth

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21 January 2018

Structural basis for the inhibition of caspase-3 by XIAP

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APRIL, a new member of the tumor necrosis factor family, modulates death ligand-induced apoptosis

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IAP suppression of apoptosis involves distinct mechanisms: the TAK1/JNK1 signaling cascade and caspase inhibition

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Melanoma inhibitor of apoptosis protein (ML-IAP) is a target for immune-mediated tumor destruction

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Bacterial lipopolysaccharide rapidly inhibits expression of C-C chemokine receptors in human monocytes

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Expression of B-cell-attracting chemokine 1 (CXCL13) by malignant lymphocytes and vascular endothelium in primary central nervous system lymphoma.

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APRIL modulates B and T cell immunity

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Caspases: enemies within

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ML-IAP, a novel inhibitor of apoptosis that is preferentially expressed in human melanomas

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Blocking of c-FLIP(L)--independent cycloheximide-induced apoptosis or Fas-mediated apoptosis by the CC chemokine receptor 9/TECK interaction

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APRIL and TALL-I and receptors BCMA and TACI: system for regulating humoral immunity

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10.1038/SJ.ONC.1208184

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Europe PubMed Central

PubMed publication ID

1 August 2017

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1 August 2017

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