cAMP and EPAC are key players in the regulation of the signal transduction pathway involved in the α-hemolysin autophagic response (Q34288762)

1 August 2017

cAMP and EPAC are key players in the regulation of the signal transduction pathway involved in the α-hemolysin autophagic response (English)

1 reference

1 August 2017

0 references

María Belén Mestre

1

1 reference

PubMed ID

22654658

1 August 2017

María Isabel Colombo

series ordinal

2

1 reference

1 August 2017

24 May 2012

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1 August 2017

1 reference

1 August 2017

8

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1 August 2017

e1002664

1 reference

1 August 2017

5

1 reference

Creative Commons Attribution 4.0 International

1 August 2017

start time

24 May 2012

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

0 references

cites work

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Development by self-digestion: molecular mechanisms and biological functions of autophagy

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cAMP effector mechanisms. Novel twists for an 'old' signaling system

6 July 2018

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6 July 2018

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6 July 2018

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6 July 2018

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Structure, function, and regulation of human cAMP-dependent protein kinases

6 July 2018

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6 July 2018

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6 July 2018

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The inositol trisphosphate receptor in the control of autophagy.

21 January 2018

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Staphylococcus aureusSubvert Autophagy for Induction of Caspase-independent Host Cell Death

21 January 2018

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Alpha-hemolysin is required for the activation of the autophagic pathway in Staphylococcus aureus-infected cells.

21 January 2018

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21 January 2018

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21 January 2018

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CREB activation and ischaemic preconditioning

21 January 2018

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Calpain-mediated cleavage of Atg5 switches autophagy to apoptosis.

21 January 2018

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The acrosomal vesicle of mouse sperm is a calcium store

21 January 2018

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Membrane-permeant Rab3A triggers acrosomal exocytosis in living human sperm.

21 January 2018

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16 Structure, function, and regulation of human cAMP-dependent protein kinases

21 January 2018

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Extracellular matrix heparan sulfate modulates endothelial cell susceptibility to Staphylococcus aureus

21 January 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/22654658

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1371/JOURNAL.PPAT.1002664

1 reference

1 August 2017

1 reference

1 August 2017

1 reference

PubMed ID

22654658