Fragile and unstable chromosomes in cancer: causes and consequences (Q34264402)

1 August 2017

title

Fragile and unstable chromosomes in cancer: causes and consequences (English)

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1 August 2017

Richards RI

1

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1 August 2017

publication date

1 June 2001

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1 August 2017

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1 August 2017

volume

17

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1 August 2017

issue

6

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1 August 2017

page(s)

339-345

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publication type of scholarly work

1 August 2017

review article

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Fragile sites still breaking

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Human chromosomal fragile site FRA16B is an amplified AT-rich minisatellite repeat

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FRA10B structure reveals common elements in repeat expansion and chromosomal fragile site genesis.

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Chromosome 3p14 homozygous deletions and sequence analysis of FRA3B.

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Sequence of the FRA3B common fragile region: Implications for the mechanism of FHIT deletion

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Fish mapping of YAC clones at human chromosomal band 7q31.2: Identification of YACS spanning FRA7G within the common region of LOH in breast and prostate cancer

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FRA7G extends over a broad region: coincidence of human endogenous retroviral sequences (HERV-H) and small polydispersed circular DNAs (spcDNA) and fragile sites

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Molecular characterization of a common fragile site (FRA7H) on human chromosome 7 by the cloning of a simian virus 40 integration site.

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The FHIT gene, spanning the chromosome 3p14.2 fragile site and renal carcinoma-associated t(3;8) breakpoint, is abnormal in digestive tract cancers

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Common chromosomal fragile site FRA16D sequence: identification of the FOR gene spanning FRA16D and homozygous deletions and translocation breakpoints in cancer cells

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The tumor-suppressor gene FHIT is involved in the regulation of apoptosis and in cell cycle control

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Muir-Torre-like syndrome in Fhit-deficient mice

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A new role for hypoxia in tumor progression: induction of fragile site triggering genomic rearrangements and formation of complex DMs and HSRs

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Expression of recessive alleles by chromosomal mechanisms in retinoblastoma

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Proximity of chromosomal loci that participate in radiation-induced rearrangements in human cells

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Translocation breakpoints in FHIT and FRA3B in both homologs of chromosome 3 in an esophageal adenocarcinoma

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7 January 2021

The Characterization of the Common Fragile Site FRA16D and Its Involvement in Multiple Myeloma Translocations

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AnFHIT tumor suppressor gene?

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Fhit, a putative tumor suppressor in humans, is a dinucleoside 5',5"'-P1,P3-triphosphate hydrolase

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The timing of cell division: Ap4A as a signal

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Replacement of Fhit in cancer cells suppresses tumorigenicity

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FHIT gene therapy prevents tumor development in Fhit-deficient mice.

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Enhanced flexibility and aphidicolin-induced DNA breaks near mammalian replication origins: implications for replicon mapping and chromosome fragility

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Role for CCG-trinucleotide repeats in the pathogenesis of chronic lymphocytic leukemia

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DNA polymerase alpha inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes

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7 January 2021

10.1016/S0168-9525(01)02303-4

Identifiers

DOI

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release_nap7qpxwjreincd7cbaj36sxhu

1 August 2017

Fatcat ID

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Fatcat

https://api.fatcat.wiki/v0/release/nap7qpxwjreincd7cbaj36sxhu

24 November 2022

mapped directly with Wikidata item

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