Pulsatility of growth hormone (GH) signalling in liver cells: role of the JAK-STAT5b pathway in GH action (Q34030528)

30 July 2017

title

Pulsatility of growth hormone (GH) signalling in liver cells: role of the JAK-STAT5b pathway in GH action (English)

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30 July 2017

2

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30 July 2017

H K Choi

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30 July 2017

publication date

1 April 2000

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Growth Hormone & I G F Research

30 July 2017

published in

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30 July 2017

volume

10 Suppl B

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30 July 2017

page(s)

S1-8

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publication type of scholarly work

30 July 2017

review article

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Mode of action of pituitary growth hormone on target cells

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Regulation of liver-specific steroid metabolizing cytochromes P450: Cholesterol 7α-hydroxylase, bile acid 6β-hydroxylase, and growth hormone-responsive steroid hormone hydroxylases

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Hypothalamo-pituitary regulation of cytochrome P-450(15) beta apoprotein levels in rat liver

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Regulation of rat hepatic cytochrome P-450: age-dependent expression, hormonal imprinting, and xenobiotic inducibility of sex-specific isoenzymes

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Intermittent plasma growth hormone triggers tyrosine phosphorylation and nuclear translocation of a liver-expressed, Stat 5-related DNA binding protein. Proposed role as an intracellular regulator of male-specific liver gene transcription

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Dimerization of the extracellular domain of the human growth hormone receptor by a single hormone molecule

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Growth Hormone, but Not Prolactin, Maintains Low-Level Activation of STAT5a and STAT5b in Female Rat Liver

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Differential Control of the Phosphorylation State of Proline-juxtaposed Serine Residues Ser725 of Stat5a and Ser730 of Stat5b in Prolactin-sensitive Cells

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Interaction of Growth Hormone-activated STATs with SH2-containing Phosphotyrosine Phosphatase SHP-1 and Nuclear JAK2 Tyrosine Kinase

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Growth Hormone Induction of Hepatic Serine Protease Inhibitor 2.1 Transcription Is Mediated by a Stat5-related Factor Binding Synergistically to Two γ-Activated Sites

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STAT 5 and NF-Y are involved in expression and growth hormone-mediated sexually dimorphic regulation of cytochrome P450 3A10/lithocholic acid 6beta-hydroxylase

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Distinctive roles of STAT5a and STAT5b in sexual dimorphism of hepatic P450 gene expression. Impact of STAT5a gene disruption

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7 January 2021

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7 January 2021

SOCS/CIS protein inhibition of growth hormone-stimulated STAT5 signaling by multiple mechanisms

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7 January 2021

The JAK-binding protein JAB inhibits Janus tyrosine kinase activity through binding in the activation loop

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7 January 2021

Requirement of tyrosine residues 333 and 338 of the growth hormone (GH) receptor for selected GH-stimulated function

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7 January 2021

Growth Hormone-Induced Tyrosyl Phosphorylation and Deoxyribonucleic Acid Binding Activity of Stat5A and Stat5B*

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Identification of growth hormone receptor (GHR) tyrosine residues required for GHR phosphorylation and JAK2 and STAT5 activation

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7 January 2021

10.1016/S1096-6374(00)80002-7

Identifiers

DOI

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30 July 2017

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