Transgenic and knockout mice to study the renin-angiotensin system and other interacting vasoactive pathways (Q34028513)

30 July 2017

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title

Transgenic and knockout mice to study the renin-angiotensin system and other interacting vasoactive pathways (English)

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30 July 2017

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Lake-Bruse KD

1

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30 July 2017

Sigmund CD

series ordinal

2

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30 July 2017

publication date

1 April 2000

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Current Hypertension Reports

30 July 2017

published in

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30 July 2017

volume

2

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30 July 2017

issue

2

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30 July 2017

page(s)

211-216

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https://scigraph.springernature.com/pub.10.1007/s11906-000-0084-1

30 July 2017

exact match

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cites work

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Cardiovascular phenotype of a mouse strain with disruption of bradykinin B2-receptor gene.

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Cyclooxygenase-2 mediates increased renal renin content induced by low-sodium diet.

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Prostaglandin synthase 2 gene disruption causes severe renal pathology in the mouse.

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Homeostasis in mice with genetically decreased angiotensinogen is primarily by an increased number of renin-producing cells

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Endothelial nitric oxide gene knockout mice: cardiac phenotypes and the effect of angiotensin-converting enzyme inhibitor on myocardial ischemia/reperfusion injury

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Absence of tubuloglomerular feedback responses in AT1A receptor-deficient mice

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Cyclooxygenase-2 inhibition decreases renin content and lowers blood pressure in a model of renovascular hypertension

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The kidney androgen-regulated protein promoter confers renal proximal tubule cell-specific and highly androgen-responsive expression on the human angiotensinogen gene in transgenic mice

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Murine double nullizygotes of the angiotensin type 1A and 1B receptor genes duplicate severe abnormal phenotypes of angiotensinogen nullizygotes

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Hypertension in mice lacking the gene for endothelial nitric oxide synthase

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Expression of neuronal type nitric oxide synthase and renin in the juxtaglomerular apparatus of angiotensin type-1a receptor gene-knockout mice

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Targeting deletion of angiotensin type 1B receptor gene in the mouse.

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Mice lacking inducible nitric oxide synthase are not resistant to lipopolysaccharide-induced death

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Highly Regulated Cell Type-restricted Expression of Human Renin in Mice Containing 140- or 160-Kilobase Pair P1 Phage Artificial Chromosome Transgenes

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Targeted inactivation of the Ren-2 gene in mice

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What we can learn from the selective manipulation of dopaminergic receptors about the pathogenesis and treatment of hypertension?

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12 December 2020

10.1007/S11906-000-0084-1

Identifiers

DOI

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Dimensions Publication ID

30 July 2017

1049158046

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