2-Deoxy-D-glucose treatment of endothelial cells induces autophagy by reactive oxygen species-mediated activation of the AMP-activated protein kinase (Q33840827)

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English	2-Deoxy-D-glucose treatment of endothelial cells induces autophagy by reactive oxygen species-mediated activation of the AMP-activated protein kinase	scientific article

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scholarly article

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Europe PubMed Central

29 July 2017

2-Deoxy-D-glucose treatment of endothelial cells induces autophagy by reactive oxygen species-mediated activation of the AMP-activated protein kinase (English)

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Europe PubMed Central

29 July 2017

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reactive oxygen species

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based on heuristic

inferred from title

author name string

Qilong Wang

1

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Europe PubMed Central

29 July 2017

Bin Liang

2

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Europe PubMed Central

29 July 2017

Najeeb A Shirwany

3

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Europe PubMed Central

29 July 2017

Ming-Hui Zou

4

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Europe PubMed Central

29 July 2017

language of work or name

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publication date

28 February 2011

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Europe PubMed Central

29 July 2017

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Europe PubMed Central

29 July 2017

6

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Europe PubMed Central

29 July 2017

2

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Europe PubMed Central

29 July 2017

e17234

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Europe PubMed Central

29 July 2017

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Creative Commons Attribution 4.0 International

28 February 2011

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April 2022 Public Data File from Crossref

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Oxidative stress and autophagy in cardiac disease, neurological disorders, aging and cancer

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AMPK in cardiovascular health and disease

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FoxO transcription factors promote autophagy in cardiomyocytes

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5 July 2018

Activation and signaling by the AMP-activated protein kinase in endothelial cells

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MUC1 oncoprotein promotes autophagy in a survival response to glucose deprivation

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Superoxide is the major reactive oxygen species regulating autophagy

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The role of autophagy in the heart.

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Resveratrol protects ROS-induced cell death by activating AMPK in H9c2 cardiac muscle cells

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Upregulation of mitochondrial uncoupling protein-2 by the AMP-activated protein kinase in endothelial cells attenuates oxidative stress in diabetes

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NADPH oxidase-derived reactive oxygen species in the regulation of endothelial phenotype

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Modulation of the activities of AMP-activated protein kinase, protein kinase B, and mammalian target of rapamycin by limiting energy availability with 2-deoxyglucose

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AMP-activated protein kinase activation as a strategy for protecting vascular endothelial function

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based on heuristic

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AMP-activated protein kinase and autophagy

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10.1371/JOURNAL.PONE.0017234

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Europe PubMed Central

29 July 2017

2011PLoSO...617234W

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24 November 2022

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Europe PubMed Central

29 July 2017

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Europe PubMed Central

29 July 2017

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