Subcellular distribution of p210(BCR-ABL) in CML cell lines and primary CD34+ CML cells (Q33308579)

25 July 2017

Subcellular distribution of p210(BCR-ABL) in CML cell lines and primary CD34+ CML cells (English)

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25 July 2017

Patel H

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25 July 2017

Marley SB

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2

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25 July 2017

Greener L

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3

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25 July 2017

Gordon MY

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4

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25 July 2017

6 December 2007

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25 July 2017

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25 July 2017

22

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3

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25 July 2017

559-571

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https://scigraph.springernature.com/pub.10.1038/sj.leu.2405057

25 July 2017

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MLL 5 protein forms intranuclear foci, and overexpression inhibits cell cycle progression

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SHP-2 phosphatase is required for hematopoietic cell transformation by Bcr-Abl

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Tyrosine 207 in CRKL is the BCR/ABL phosphorylation site

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Structural requirements for function of the Crkl adapter protein in fibroblasts and hematopoietic cells

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Abl protein-tyrosine kinase selects the Crk adapter as a substrate using SH3-binding sites

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Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells

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BCR-ABL oncoprotein is expressed by platelets from CML patients and associated with a special pattern of CrkL phosphorylation

21 January 2018

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An in vivo and in vitro comparison of the effects of b2-a2 and b3-a2 p210BCR-ABL splice variants on murine 32D cells

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7 January 2021

Bcr-Abl oncoproteins bind directly to activators of the Ras signalling pathway

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The tyrosine kinase inhibitor STI571, like interferon-alpha, preferentially reduces the capacity for amplification of granulocyte-macrophage progenitors from patients with chronic myeloid leukemia

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Effects of the Tyrosine Kinase Inhibitor Imatinib Mesylate on a Bcr-Abl-Positive Cell Line: Suppression of Autonomous Cell Growth but No Effect on Decreased Adhesive Property and Morphological Changes

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7 January 2021

Identifiers

DOI

10.1038/SJ.LEU.2405057

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Dimensions Publication ID

25 July 2017

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