Contemporary avian influenza A virus subtype H1, H6, H7, H10, and H15 hemagglutinin genes encode a mammalian virulence factor similar to the 1918 pandemic virus H1 hemagglutinin. (Q30368757)

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English	Contemporary avian influenza A virus subtype H1, H6, H7, H10, and H15 hemagglutinin genes encode a mammalian virulence factor similar to the 1918 pandemic virus H1 hemagglutinin.	scientific article

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scholarly article

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14 October 2017

Contemporary avian influenza A virus subtype H1, H6, H7, H10, and H15 hemagglutinin genes encode a mammalian virulence factor similar to the 1918 pandemic virus H1 hemagglutinin. (English)

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14 October 2017

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avian influenza

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influenza A virus

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influenza A virus

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Jeffery Taubenberger

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Jeffery K Taubenberger

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14 October 2017

Kathie-Anne Walters

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Kathie-Anne Walters

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Louis M Schwartzman

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Louis M Schwartzman

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Daniel S Chertow

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Daniel S Chertow

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Kelsey Scherler

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Kelsey Scherler

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Li Qi

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Lindsey M Pujanauski

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A Sally Davis

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David Baxter

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Kevan L Hartshorn

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Richard D Slemons

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John C Kash

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18 November 2014

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14 October 2017

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14 October 2017

e02116

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Europe PubMed Central

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14 October 2017

Circulating avian influenza viruses closely related to the 1918 virus have pandemic potential.

1 reference

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Clinical and epidemiological characteristics of a fatal case of avian influenza A H10N8 virus infection: a descriptive study.

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H6 influenza viruses pose a potential threat to human health

1 reference

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Human infection with avian influenza A H6N1 virus: an epidemiological analysis

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Emergence in China of human disease due to avian influenza A(H10N8)--cause for concern?

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Treatment with the reactive oxygen species scavenger EUK-207 reduces lung damage and increases survival during 1918 influenza virus infection in mice

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Outbreak of variant influenza A(H3N2) virus in the United States.

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H7N9 avian influenza A virus and the perpetual challenge of potential human pandemicity

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Genomic signature and protein sequence analysis of a novel influenza A (H7N9) virus that causes an outbreak in humans in China

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Human infections with the emerging avian influenza A H7N9 virus from wet market poultry: clinical analysis and characterisation of viral genome

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Changes in microRNA and mRNA expression with differentiation of human bronchial epithelial cells

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Human infection with a novel avian-origin influenza A (H7N9) virus

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High-throughput RNA sequencing of a formalin-fixed, paraffin-embedded autopsy lung tissue sample from the 1918 influenza pandemic

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Reconstruction of the 1918 influenza virus: unexpected rewards from the past

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The potential of avian H1N1 influenza A viruses to replicate and cause disease in mammalian models

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Engineering H5N1 avian influenza viruses to study human adaptation

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Analysis by single-gene reassortment demonstrates that the 1918 influenza virus is functionally compatible with a low-pathogenicity avian influenza virus in mice

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Emergence of mammalian species-infectious and -pathogenic avian influenza H6N5 virus with no evidence of adaptation

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Autopsy series of 68 cases dying before and during the 1918 influenza pandemic peak

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Insights on influenza pathogenesis from the grave

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Reversion of PB2-627E to -627K during replication of an H5N1 Clade 2.2 virus in mammalian hosts depends on the origin of the nucleoprotein

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Pandemic influenza: certain uncertainties

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The ability of pandemic influenza virus hemagglutinins to induce lower respiratory pathology is associated with decreased surfactant protein D binding

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Pathogenicity of swine influenza viruses possessing an avian or swine-origin PB2 polymerase gene evaluated in mouse and pig models

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Full genome comparison and characterization of avian H10 viruses with different pathogenicity in Mink (Mustela vison) reveals genetic and functional differences in the non-structural gene.

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Influenza virus evolution, host adaptation, and pandemic formation

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Role of surfactant protein A and D (SP-A and SP-D) in human antiviral host defense

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An early 'classical' swine H1N1 influenza virus shows similar pathogenicity to the 1918 pandemic virus in ferrets and mice.

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A single-amino-acid substitution in a polymerase protein of an H5N1 influenza virus is associated with systemic infection and impaired T-cell activation in mice

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Avian influenza viruses in humans.

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Different evolutionary trajectories of European avian-like and classical swine H1N1 influenza A viruses.

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Cross-Species Virus Transmission and the Emergence of New Epidemic Diseases

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Avian influenza h6 viruses productively infect and cause illness in mice and ferrets

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H5N1 and 1918 pandemic influenza virus infection results in early and excessive infiltration of macrophages and neutrophils in the lungs of mice.

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Single gene reassortants identify a critical role for PB1, HA, and NA in the high virulence of the 1918 pandemic influenza virus

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The pathology of influenza virus infections

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The Mx1 gene protects mice against the pandemic 1918 and highly lethal human H5N1 influenza viruses.

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Aberrant innate immune response in lethal infection of macaques with the 1918 influenza virus

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A single gene determines the host range of influenza virus

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4251996

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Poultry carrying H9N2 act as incubators for novel human avian influenza viruses.

1 reference

https://api.crossref.org/works/10.1128%2FMBIO.02116-14

21 January 2018

Influenza revisited

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21 January 2018

TM4 microarray software suite

1 reference

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21 January 2018

Identifiers

10.1128/MBIO.02116-14

1 reference

Europe PubMed Central

PMC publication ID

14 October 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 October 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

14 October 2017

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