Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis (Q29620506)

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23 December 2019

title

Interleukin-22, a T(H)17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis (English)

2 references

PubMed

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30 April 2017

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23 December 2019

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0 references

2

1 reference

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23 December 2019

Wenjun Ouyang

7

1 reference

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23 December 2019

Jeffrey Eastham-Anderson

5

object named as

Jeffrey Eastham-Anderson

1 reference

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23 December 2019

author name string

Yan Zheng

1

1 reference

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23 December 2019

Patricia Valdez

3

1 reference

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23 December 2019

Ian Kasman

4

1 reference

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23 December 2019

Jianfeng Wu

6

1 reference

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23 December 2019

language of work or name

1 reference

30 April 2017

publication date

24 December 2006

1 reference

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23 December 2019

2 references

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30 April 2017

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23 December 2019

volume

445

2 references

PubMed

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30 April 2017

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23 December 2019

page(s)

648-651

1 reference

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23 December 2019

issue

7128

2 references

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30 April 2017

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Recent insights into the immunopathogenesis of psoriasis provide new therapeutic opportunities

23 December 2019

cites work

1 reference

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7 January 2021

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

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Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

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Increased expression of interleukin 23 p19 and p40 in lesional skin of patients with psoriasis vulgaris

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IL-22, a Th1 cytokine that targets the pancreas and select other peripheral tissues

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IL-22 increases the innate immunity of tissues

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IL-22 inhibits epidermal differentiation and induces proinflammatory gene expression and migration of human keratinocytes

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IL-22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: a potential role in psoriasis

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7 January 2021

Interleukin (IL)-22 and IL-17 are coexpressed by Th17 cells and cooperatively enhance expression of antimicrobial peptides

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7 January 2021

Understanding the IL-23-IL-17 immune pathway

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A receptor for the heterodimeric cytokine IL-23 is composed of IL-12Rbeta1 and a novel cytokine receptor subunit, IL-23R

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7 January 2021

Th17: an effector CD4 T cell lineage with regulatory T cell ties

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TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

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Transforming growth factor-beta induces development of the T(H)17 lineage

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Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

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IL-23 production by cosecretion of endogenous p19 and transgenic p40 in keratin 14/p40 transgenic mice: evidence for enhanced cutaneous immunity

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Inflammatory skin disease in K14/p40 transgenic mice: evidence for interleukin-12-like activities of p40.

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7 January 2021

IL-23 stimulates epidermal hyperplasia via TNF and IL-20R2-dependent mechanisms with implications for psoriasis pathogenesis

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7 January 2021

Interleukin-17 and interferon-gamma synergize in the enhancement of proinflammatory cytokine production by human keratinocytes.

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7 January 2021

Stat3 links activated keratinocytes and immunocytes required for development of psoriasis in a novel transgenic mouse model

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7 January 2021

Use of monospecific antisera and cRNA probes to localize the major changes in keratin expression during normal and abnormal epidermal differentiation

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7 January 2021

Interleukin-1 induces transcription of keratin K6 in human epidermal keratinocytes

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7 January 2021

Epidermal growth factor and transforming growth factor alpha specifically induce the activation- and hyperproliferation-associated keratins 6 and 16.

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Increase in TNF-alpha and inducible nitric oxide synthase-expressing dendritic cells in psoriasis and reduction with efalizumab (anti-CD11a)

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7 January 2021

Blockade of T lymphocyte costimulation with cytotoxic T lymphocyte-associated antigen 4-immunoglobulin (CTLA4Ig) reverses the cellular pathology of psoriatic plaques, including the activation of keratinocytes, dendritic cells, and endothelial cells

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7 January 2021

Characterization of dermal dendritic cells in psoriasis. Autostimulation of T lymphocytes and induction of Th1 type cytokines

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7 January 2021

The majority of epidermal T cells in Psoriasis vulgaris lesions can produce type 1 cytokines, interferon-gamma, interleukin-2, and tumor necrosis factor-alpha, defining TC1 (cytotoxic T lymphocyte) and TH1 effector populations: a type 1 differentiat

1 reference

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7 January 2021

T cells involved in psoriasis vulgaris belong to the Th1 subset

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7 January 2021

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=17187052

Identifiers

3 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017

685060

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17187052%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

23 December 2019

1007842569

0 references

685060

1 reference

Consolidated OpenCitations Corpus – April 2017

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=17187052

3 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017

685060