Multiply attenuated lentiviral vector achieves efficient gene delivery in vivo (Q29616612)

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30 April 2017

4

L. Naldini

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17 November 2024

author name string

R. Zufferey

1

1 reference

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17 November 2024

D. Nagy

2

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17 November 2024

R. J. Mandel

3

1 reference

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D. Trono

5

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publication date

September 1997

1 reference

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30 April 2017

language of work or name

1 reference

http://www.nature.com/articles/nbt0997-871

30 April 2017

work available at URL

1 reference

https://api.crossref.org/works/10.1038/NBT0997-871

http://www.nature.com/articles/nbt0997-871.pdf

17 November 2024

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17 November 2024

1 reference

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30 April 2017

volume

15

1 reference

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issue

9

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30 April 2017

page(s)

871-5

1 reference

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The basic science of gene therapy

30 April 2017

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The nuclear localization signal of the matrix protein of human immunodeficiency virus type 1 allows the establishment of infection in macrophages and quiescent T lymphocytes

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Efficient transfer, integration, and sustained long-term expression of the transgene in adult rat brains injected with a lentiviral vector

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Role of vif in replication of human immunodeficiency virus type 1 in CD4+ T lymphocytes

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The human immunodeficiency virus type 1-specific protein vpu is required for efficient virus maturation and release

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Vpu protein of human immunodeficiency virus type 1 enhances the release of capsids produced by gag gene constructs of widely divergent retroviruses

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The human immunodeficiency virus-1 nef gene product: a positive factor for viral infection and replication in primary lymphocytes and macrophages

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The importance of nef in the induction of human immunodeficiency virus type 1 replication from primary quiescent CD4 lymphocytes.

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Nef stimulates human immunodeficiency virus type 1 proviral DNA synthesis

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The growth advantage conferred by HIV-1 nef is determined at the level of viral DNA formation and is independent of CD4 downregulation.

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Pseudotyping human immunodeficiency virus type 1 (HIV-1) by the glycoprotein of vesicular stomatitis virus targets HIV-1 entry to an endocytic pathway and suppresses both the requirement for Nef and the sensitivity to cyclosporin A

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=9306402

Identifiers

2 references

30 April 2017

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

512172

Dimensions publication ID

1006020704

0 references

OpenCitations bibliographic resource ID

512172

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

512172