Opposing roles for NF-kappa B/Rel factors p65 and c-Rel in the modulation of neuron survival elicited by glutamate and interleukin-1beta (Q28581717)

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8 December 2019

title

Opposing roles for NF-kappa B/Rel factors p65 and c-Rel in the modulation of neuron survival elicited by glutamate and interleukin-1beta (English)

2 references

31 January 2017

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8 December 2019

0 references

1 reference

1

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8 December 2019

PierFranco Spano

series ordinal

7

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3

Flora Boroni

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4

Marina Benarese

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2

Francesca Goffi

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31 January 2017

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6

Hsiou-Chi Liou

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8 December 2019

author name string

Scott E Perkins

series ordinal

5

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8 December 2019

language of work or name

English

0 references

publication date

23 March 2002

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Journal of Biological Chemistry

8 December 2019

published in

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volume

277

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8 December 2019

issue

23

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8 December 2019

page(s)

20717-20723

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Regulation of the NF-kappa B/rel transcription factor and I kappa B inhibitor system

8 December 2019

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Possible role of NF-kappaB and p53 in the glutamate-induced pro-apoptotic neuronal pathway

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Enhancement of immunoreactivity for NF-kappa B in human cerebral infarctions.

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Global ischemia activates nuclear factor-kappa B in forebrain neurons of rats.

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NF-kappaB is activated and promotes cell death in focal cerebral ischemia.

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Transcription factor NF-kappaB is activated in primary neurons by amyloid beta peptides and in neurons surrounding early plaques from patients with Alzheimer disease

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Cytokine-induced nuclear factor kappa B activation promotes the survival of developing neurons.

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Activation of NF-kappaB protects hippocampal neurons against oxidative stress-induced apoptosis: evidence for induction of manganese superoxide dismutase and suppression of peroxynitrite production and protein tyrosine nitration.

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Tumor necrosis factor induces Bcl-2 and Bcl-x expression through NFkappaB activation in primary hippocampal neurons

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Inhibition of NF-kappaB potentiates amyloid beta-mediated neuronal apoptosis

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Neuroprotection by aspirin and sodium salicylate through blockade of NF-kappaB activation.

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NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses

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I kappa B-independent control of NF-kappa B activity by modulatory phosphorylations

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Achieving transcriptional specificity with NF-kappa B.

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The ability of CD40L, but not lipopolysaccharide, to initiate immunoglobulin switching to immunoglobulin G1 is explained by differential induction of NF-kappaB/Rel proteins

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Manipulation of distinct NFkappaB proteins alters interleukin-1beta-induced human rheumatoid synovial fibroblast prostaglandin E2 formation

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Interleukin-1beta and glutamate activate the NF-kappaB/Rel binding site from the regulatory region of the amyloid precursor protein gene in primary neuronal cultures.

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Excitatory amino acids as a final common pathway for neurologic disorders.

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A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells

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IkappaB family members function by different mechanisms

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Insulin-like growth factor-1-mediated neuroprotection against oxidative stress is associated with activation of nuclear factor kappaB.

21 January 2018

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The IKK complex: an integrator of all signals that activate NF-kappaB?

21 January 2018

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Mechanism of I kappa B alpha binding to NF-kappa B dimers

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Distinct roles of the Ikappa B kinase alpha and beta subunits in liberating nuclear factor kappa B (NF-kappa B) from Ikappa B and in phosphorylating the p65 subunit of NF-kappa B.

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Rel-dependent induction of A1 transcription is required to protect B cells from antigen receptor ligation-induced apoptosis

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The prosurvival Bcl-2 homolog Bfl-1/A1 is a direct transcriptional target of NF-kappaB that blocks TNFalpha-induced apoptosis

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Activation of Multiple Metabotropic Glutamate Receptor Subtypes Prevents NMDA-induced Excitotoxicity in Rat Hippocampal Slices

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Identifiers

DOI

10.1074/JBC.M201014200

2 references

Consolidated OpenCitations Corpus – April 2017

2605631

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8 December 2019

2605631

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

2 references

2605631