Response to myocardial ischemia/reperfusion injury involves Bnip3 and autophagy (Q28576035)

2

1 reference

31 January 2017

M. R. Sayen

4

1 reference

31 January 2017

M. Jinno

5

1 reference

31 January 2017

L. A. Kirshenbaum

6

1 reference

31 January 2017

R. A. Gottlieb

7

1 reference

31 January 2017

A. B. Gustafsson

Cell Death & Differentiation

8

1 reference

31 January 2017

1 January 2007

1 reference

31 January 2017

1 reference

31 January 2017

14

1 reference

31 January 2017

146–157

1 reference

31 January 2017

1

1 reference

31 January 2017

Acute myocardial infarction in humans is associated with activation of programmed myocyte cell death in the surviving portion of the heart

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Apoptosis in myocytes in end-stage heart failure

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

BID: a novel BH3 domain-only death agonist

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Adenovirus E1B-19K/BCL-2 interacting protein BNIP3 contains a BH3 domain and a mitochondrial targeting sequence

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

BNIP3 heterodimerizes with Bcl-2/Bcl-X(L) and induces cell death independent of a Bcl-2 homology 3 (BH3) domain at both mitochondrial and nonmitochondrial sites

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

The E1B 19K/Bcl-2-binding protein Nip3 is a dimeric mitochondrial protein that activates apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Inducible expression of BNIP3 provokes mitochondrial defects and hypoxia-mediated cell death of ventricular myocytes.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Expression of the gene encoding the proapoptotic Nip3 protein is induced by hypoxia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Hypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Pivotal role of the cell death factor BNIP3 in ceramide-induced autophagic cell death in malignant glioma cells.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Induction of autophagy and inhibition of tumorigenesis by beclin 1

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Autophagic degeneration as a possible mechanism of myocardial cell death in dilated cardiomyopathy

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Diversity in the mechanisms of neuronal cell death.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

TAT protein transduction into isolated perfused hearts: TAT-apoptosis repressor with caspase recruitment domain is cardioprotective

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Apoptosis repressor with caspase recruitment domain protects against cell death by interfering with Bax activation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Bid is cleaved by calpain to an active fragment in vitro and during myocardial ischemia/reperfusion.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Autophagy in chronically ischemic myocardium.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Studies on the mechanisms of autophagy: formation of the autophagic vacuole

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Inhibition of macroautophagy triggers apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Growth factor regulation of autophagy and cell survival in the absence of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Dissection of autophagosome formation using Apg5-deficient mouse embryonic stem cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Essential roles of Atg5 and FADD in autophagic cell death: dissection of autophagic cell death into vacuole formation and cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Effect of vacuolar proton ATPase on pHi, Ca2+, and apoptosis in neonatal cardiomyocytes during metabolic inhibition/recovery

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Enhanced expression of p53 and apoptosis induced by blockade of the vacuolar proton ATPase in cardiomyocytes

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Mechanism of preconditioning. Ionic alterations

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

The mitochondrial permeability transition initiates autophagy in rat hepatocytes.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Mitochondria are selectively eliminated from eukaryotic cells after blockade of caspases during apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Protection by Cyclosporin A of ischemia/reperfusion-induced damage in isolated rat hearts.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

alpha 1-adrenergic agonists precondition rabbit ischemic myocardium independent of adenosine by direct activation of protein kinase C

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

Calcineurin transgenic mice have mitochondrial dysfunction and elevated superoxide production

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

HL-1 cells: a cardiac muscle cell line that contracts and retains phenotypic characteristics of the adult cardiomyocyte

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

A monomeric red fluorescent protein

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021

YOPRO-1 permits cytofluorometric analysis of programmed cell death (apoptosis) without interfering with cell viability.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401936

7 January 2021