Krüppel-like factor 4 inhibits proliferation by platelet-derived growth factor receptor beta-mediated, not by retinoic acid receptor alpha-mediated, phosphatidylinositol 3-kinase and ERK signaling in vascular smooth muscle cells (Q28566073)

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English	Krüppel-like factor 4 inhibits proliferation by platelet-derived growth factor receptor beta-mediated, not by retinoic acid receptor alpha-mediated, phosphatidylinositol 3-kinase and ERK signaling in vascular smooth muscle cells	scientific journal article

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31 January 2017

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31 January 2020

Krüppel-like factor 4 inhibits proliferation by platelet-derived growth factor receptor beta-mediated, not by retinoic acid receptor alpha-mediated, phosphatidylinositol 3-kinase and ERK signaling in vascular smooth muscle cells (English)

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Bin Zheng

1

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Mei Han

2

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Xin-hua Zhang

4

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Fang Meng

5

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Sui-bing Miao

6

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Ming He

7

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Xin-ming Zhao

8

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31 January 2020

Jin-kun Wen

9

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16 June 2009

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31 January 2020

Journal of Biological Chemistry

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284

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34

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22773-22785

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31 January 2020

Synthetic retinoid Am80 inhibits interaction of KLF5 with RAR alpha through inducing KLF5 dephosphorylation mediated by the PI3K/Akt signaling in vascular smooth muscle cells

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The diverse functions of Krüppel-like factors 4 and 5 in epithelial biology and pathobiology

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Krüppel-like factors 4 and 5: the yin and yang regulators of cellular proliferation

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Retinoic acid-induced tissue transglutaminase and apoptosis in vascular smooth muscle cells

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Human heart LIM protein has transcription activation ability related to LIM domain 1.

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Roscovitine inhibits ERK1/2 activation induced by angiotensin II in vascular smooth muscle cells

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Multiple repressor pathways contribute to phenotypic switching of vascular smooth muscle cells

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Akt down-regulates ERK1/2 nuclear localization and angiotensin II-induced cell proliferation through PEA-15.

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Interferon-gamma induces Fas trafficking and sensitization to apoptosis in vascular smooth muscle cells via a PI3K- and Akt-dependent mechanism

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Control of SRF binding to CArG box chromatin regulates smooth muscle gene expression in vivo.

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Emerging role of KLF4 in human gastrointestinal cancer

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Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages

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Vascular implications of the Krüppel-like family of transcription factors

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hhLIM protein is involved in cardiac hypertrophy

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Krüppel-like factor 4 (KLF4/GKLF) is a target of bone morphogenetic proteins and transforming growth factor beta 1 in the regulation of vascular smooth muscle cell phenotype.

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3 June 2018

Synthetic retinoid Am80 suppresses smooth muscle phenotypic modulation and in-stent neointima formation by inhibiting KLF5.

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10.1074/JBC.M109.026989

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31 January 2017

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31 January 2020

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31 January 2020

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31 January 2017

Europe PubMed Central

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31 January 2020

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