Mesoangioblast stem cells ameliorate muscle function in dystrophic dogs (Q28274324)

Giuseppe D'Antona

3

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Yvan Torrente

Yvan Torrente

17

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Beatriz G. Gálvez

Beatriz G Galvez

9

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Maurilio Sampaolesi

Maurilio Sampaolesi

1

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Laura Perani

Laura Perani

11

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Claudio Bordignon

18

Claudio Bordignon

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author name string

Stephane Blot

2

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Nicolas Granger

4

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Rossana Tonlorenzi

5

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Anna Innocenzi

6

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Paolo Mognol

7

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Jean-Lauren Thibaud

8

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Ines Barthélémy

10

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Maria Guttinger

13

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Orietta Pansarasa

14

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Chiara Rinaldi

15

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M Gabriella Cusella De Angelis

16

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Roberto Bottinelli

19

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Giulio Cossu

20

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language of work or name

English

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publication date

30 November 2006

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published in

Nature

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volume

444

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issue

7119

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page(s)

574-9

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cites work

The muscular dystrophies

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New therapies for muscular dystrophy: cautious optimism

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Identification of a novel population of muscle stem cells in mice: potential for muscle regeneration

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Cell Therapy of -Sarcoglycan Null Dystrophic Mice Through Intra-Arterial Delivery of Mesoangioblasts

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Systemic delivery of human microdystrophin to regenerating mouse dystrophic muscle by muscle progenitor cells

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Human circulating AC133(+) stem cells restore dystrophin expression and ameliorate function in dystrophic skeletal muscle

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Transplantation of a multipotent cell population from human adipose tissue induces dystrophin expression in the immunocompetent mdx mouse

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Bone marrow stromal cells generate muscle cells and repair muscle degeneration.

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Muscular dystrophy in a litter of golden retriever dogs

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An error in dystrophin mRNA processing in golden retriever muscular dystrophy, an animal homologue of Duchenne muscular dystrophy

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Modular flexibility of dystrophin: implications for gene therapy of Duchenne muscular dystrophy

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Complete repair of dystrophic skeletal muscle by mesoangioblasts with enhanced migration ability

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The cranial sartorius muscle undergoes true hypertrophy in dogs with golden retriever muscular dystrophy

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Mesoangioblasts, Vessel-Associated Multipotent Stem Cells, Repair the Infarcted Heart by Multiple Cellular Mechanisms

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Skinned single fibers from normal and dystrophin-deficient dogs incur comparable stretch-induced force deficits

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Regulation of class I MHC expression in skeletal muscle: deleterious effect of aberrant expression on myogenesis

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Bone marrow mesenchymal stem cells inhibit the response of naive and memory antigen-specific T cells to their cognate peptide.

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Visualizing the generation of memory CD4 T cells in the whole body

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Hematopoietic stem cell transplantation does not restore dystrophin expression in Duchenne muscular dystrophy dogs

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Gene therapy research for Duchenne and Becker muscular dystrophies

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Use of the dog model for Duchenne muscular dystrophy in gene therapy trials.

1 reference

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Dystrophic phenotype of canine X-linked muscular dystrophy is mitigated by adenovirus-mediated utrophin gene transfer.

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Clinical islet transplantation: advances and immunological challenges

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Identifiers

DOI

10.1038/NATURE05282

1 reference

Consolidated OpenCitations Corpus – April 2017

Dimensions Publication ID

1904543

1011740155

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1904543

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference