Differential patterns of regional c-Fos induction in the rat brain by amphetamine and the novel wakefulness-promoting agent modafinil (Q28265143)

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S A Dennis

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M S Miller

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P C Contreras

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R V Bhat

Effect of modafinil and amphetamine on the rat catecholaminergic neuron activity

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language of work or name

English

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publication date

30 January 1998

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published in

Neuroscience Letters

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volume

241

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issue

2-3

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page(s)

95-8

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cites work

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D1 dopamine receptor activation of multiple transcription factor genes in rat striatum

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Lack of pre-synaptic dopaminergic involvement in modafinil activity in anaesthetized mice: in vivo voltammetry studies.

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Brain regional substrates for the actions of the novel wake-promoting agent modafinil in the rat: comparison with amphetamine.

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The vigilance promoting drug modafinil decreases GABA release in the medial preoptic area and in the posterior hypothalamus of the awake rat: possible involvement of the serotonergic 5-HT3 receptor.

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Amphetamine and cocaine induce drug-specific activation of the c-fos gene in striosome-matrix compartments and limbic subdivisions of the striatum

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Amygdaloid projections to subcortical structures within the basal forebrain and brainstem in the rat and cat

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Electroencephalographic effects of modafinil, an alpha-1-adrenergic psychostimulant, on the sleep of rhesus monkeys.

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Potential brain neuronal targets for amphetamine-, methylphenidate-, and modafinil-induced wakefulness, evidenced by c-fos immunocytochemistry in the cat.

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Activation of ventrolateral preoptic neurons during sleep

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Awakening properties of modafinil without paradoxical sleep rebound: comparative study with amphetamine in the rat.

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Dopamine agonists and stress produce different patterns of Fos-like immunoreactivity in the lateral habenula

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Ultrastructural evidence of amygdalofugal axons terminating on cholinergic cells of the rostral forebrain.

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