Chemokine receptor utilization and macrophage signaling by human immunodeficiency virus type 1 gp120: Implications for neuropathogenesis (Q28246686)

Europe PubMed Central

title

Chemokine receptor utilization and macrophage signaling by human immunodeficiency virus type 1 gp120: Implications for neuropathogenesis (English)

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9 April 2017

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Ronald Collman

5

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ORCID Public Data File 2021

author name string

Yanjie Yi

1

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9 April 2017

ChuHee Lee

2

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9 April 2017

Qing-Hua Liu

3

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9 April 2017

Bruce D Freedman

4

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Ronald G Collman

5

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Bruce Freedman

4

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9 April 2017

language of work or name

English

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publication date

2004

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9 April 2017

published in

Journal of NeuroVirology

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9 April 2017

volume

10 Suppl 1

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10

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9 April 2017

issue

1

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9 April 2017

page(s)

91-6

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91-96

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Beta-chemokines MCP-1 and RANTES are selectively increased in cerebrospinal fluid of patients with human immunodeficiency virus-associated dementia

9 April 2017

cites work

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Early activation of mitogen-activated protein kinase kinase, extracellular signal-regulated kinase, p38 mitogen-activated protein kinase, and c-Jun N-terminal kinase in response to binding of simian immunodeficiency virus to Jurkat T cells expressin

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HIV-1 gp120 stimulates the production of beta-chemokines in human peripheral blood monocytes through a CD4-independent mechanism

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Elevated cerebrospinal fluid levels of monocyte chemotactic protein-1 correlate with HIV-1 encephalitis and local viral replication.

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HIV-1 induces tumour necrosis factor and IL-1 gene expression in primary human macrophages independent of productive infection

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Macrophage-tropic HIV and SIV envelope proteins induce a signal through the CCR5 chemokine receptor

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HIV-1 gp120 and chemokine activation of Pyk2 and mitogen-activated protein kinases in primary macrophages mediated by calcium-dependent, pertussis toxin-insensitive chemokine receptor signaling

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Signal transduction due to HIV-1 envelope interactions with chemokine receptors CXCR4 or CCR5.

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Lymphotropic virions affect chemokine receptor-mediated neural signaling and apoptosis: implications for human immunodeficiency virus type 1-associated dementia

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An unusual syncytia-inducing human immunodeficiency virus type 1 primary isolate from the central nervous system that is restricted to CXCR4, replicates efficiently in macrophages, and induces neuronal apoptosis

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Immunocytochemical quantitation of human immunodeficiency virus in the brain: correlations with dementia

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Neuronal apoptosis induced by HIV-1 gp120 and the chemokine SDF-1 alpha is mediated by the chemokine receptor CXCR4.

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CXCR-4 (Fusin), a co-receptor for the type 1 human immunodeficiency virus (HIV-1), is expressed in the human brain in a variety of cell types, including microglia and neurons.

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Protein tyrosine kinase PYK2 involved in Ca(2+)-induced regulation of ion channel and MAP kinase functions

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CXCR-4 is expressed by primary macrophages and supports CCR5-independent infection by dual-tropic but not T-tropic isolates of human immunodeficiency virus type 1

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Development of a human neuronal cell model for human immunodeficiency virus (HIV)-infected macrophage-induced neurotoxicity: apoptosis induced by HIV type 1 primary isolates and evidence for involvement of the Bcl-2/Bcl-xL-sensitive intrinsic apopto

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Identifiers

DOI

10.1080/753312758

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