Primary Sjögren's syndrome and deficiency of ICA69 (Q28207888)

2

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Roy Cheung

3

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Hubert Tsui

4

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Aihua Song

5

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Roger Gaedigk

6

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Daniel Winer

7

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Anastasia Sampson

8

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Colin McKerlie

9

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Arthur Bookman

10

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H-Michael Dosch

Update in Sjögren syndrome

11

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language of work or name

English

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publication date

5 October 2002

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published in

The Lancet

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volume

360

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issue

9339

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page(s)

1063-9

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cites work

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Role of viruses in systemic lupus erythematosus and Sjögren syndrome

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Current issues in the diagnosis and treatment of Sjögren's syndrome

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Anti-Ro (SS-A) and anti-La (SS-B) in patients with Sjögren's syndrome

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SS-56, a novel cellular target of autoantibody responses in Sjögren syndrome and systemic lupus erythematosus

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Identification of alpha-fodrin as a candidate autoantigen in primary Sjögren's syndrome

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Autoantibodies to the amino-terminal fragment of beta-fodrin expressed in glandular epithelial cells in patients with Sjögren's syndrome

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Transfer of human serum IgG to nonobese diabetic Igmu null mice reveals a role for autoantibodies in the loss of secretory function of exocrine tissues in Sjögren's syndrome

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Utilization of the non-obese diabetic (NOD) mouse as an animal model for the study of secondary Sjögren's syndrome

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Sjögren's syndrome in MRL/l and MRL/n mice

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A novel NOD-derived murine model of primary Sjögren's syndrome

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Loss of self-tolerance to ICA69 in nonobese diabetic mice

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The diabetes autoantigen ICA69 and its Caenorhabditis elegans homologue, ric-19, are conserved regulators of neuroendocrine secretion

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ICA69(null) nonobese diabetic mice develop diabetes, but resist disease acceleration by cyclophosphamide.

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Type I diabetes and multiple sclerosis patients target islet plus central nervous system autoantigens; nonimmunized nonobese diabetic mice can develop autoimmune encephalitis.

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B lymphocytes are essential for the initiation of T cell-mediated autoimmune diabetes: analysis of a new "speed congenic" stock of NOD.Ig mu null mice

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Peptide dose, MHC affinity, and target self-antigen expression are critical for effective immunotherapy of nonobese diabetic mouse prediabetes

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Measurement of T-cell autoreactivity in autoimmune diabetes

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Male gonadal environment paradoxically promotes dacryoadenitis in nonobese diabetic mice.

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Evidence that a peptide spanning the B-C junction of proinsulin is an early Autoantigen epitope in the pathogenesis of type 1 diabetes

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Subacute sensory neuronopathy secondary to dorsal root ganglionitis in primary Sjögren's syndrome

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Sjögren's syndrome association with HLA-Dw3

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Anti-120-kDa alpha-fodrin immune response with Th1-cytokine profile in the NOD mouse model of Sjögren's syndrome

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Elevated proapoptotic Bax and caspase 3 activation in the NOD.scid model of Sjögren's syndrome

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Genetically programmed development of salivary gland abnormalities in the NOD (nonobese diabetic)-scid mouse in the absence of detectable lymphocytic infiltration: a potential trigger for sialoadenitis of NOD mice

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